39584
3-Sulfopyruvic acid lithium salt hydrate
≥97.0% (TLC)
Synonym(s):
2-Oxo-3-sulfopropanoic acid lithium salt hydrate, Lithium 3-sulfopyruvate hydrate
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About This Item
Empirical Formula (Hill Notation):
C3H4O6S
Molecular Weight:
168.13
MDL number:
UNSPSC Code:
41116107
PubChem Substance ID:
NACRES:
NA.24
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grade
analytical standard
Quality Level
assay
≥97.0% (TLC)
impurities
≤15% water
format
neat
storage temp.
2-8°C
SMILES string
O=C(C(O)=O)CS(=O)(O)=O
InChI
1S/C3H4O6S/c4-2(3(5)6)1-10(7,8)9/h1H2,(H,5,6)(H,7,8,9)
InChI key
BUTHMSUEBYPMKJ-UHFFFAOYSA-N
Biochem/physiol Actions
3-Sulfopyruvic acid is the product of the transamination of cysteinesulfonate in a reaction catalyzed by aspartate amino- transferase. 3-Sulfopyruvic acid is stable and is reduced by malate dehydrogenase to beta-sulfolactate, which is excreted in the urine. Cysteinesulfonate, 3-sulfo- pyruvic acid, and beta-sulfolactate are reversibly interconverted in vivo.
Storage Class
13 - Non Combustible Solids
wgk_germany
WGK 3
flash_point_f
Not applicable
flash_point_c
Not applicable
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C L Weinstein et al.
The Journal of biological chemistry, 263(8), 3735-3743 (1988-03-15)
L-Cysteinesulfonate (L-cysteate) is present in plasma, urine, and tissues in concentrations comparable to that of L-cysteinesulfinate, the primary oxidative metabolite of L-cysteine. Although cysteinesulfonate is known to be decarboxylated to taurine by cysteinesulfinate decarboxylase, the occurrence and importance of other
Mammalian sulfur amino acid metabolism: an overview.
O W Griffith
Methods in enzymology, 143, 366-376 (1987-01-01)
Three rare diseases in one Sib pair: RAI1, PCK1, GRIN2B mutations associated with Smith-Magenis Syndrome, cytosolic PEPCK deficiency and NMDA receptor glutamate insensitivity.
Adams DR, Yuan H, Holyoak T, et al.
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Signaling And Metabolism In Cancer: Endocrine Pancreas Deficiency And Hybrid Anabolism - Catabolism, Drugs That Undo The Process
Israel, M.
Cancer Therapy, 10, 1-1 (2014)
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Multiple models of human metabolism have been reconstructed, but each represents only a subset of our knowledge. Here we describe Recon 2, a community-driven, consensus 'metabolic reconstruction', which is the most comprehensive representation of human metabolism that is applicable to
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