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48505

Supelco

Carbon tetrachloride

analytical standard

Synonym(s):

Tetrachloromethane

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About This Item

Empirical Formula (Hill Notation):
CCl4
CAS Number:
56-23-5
Molecular Weight:
153.82
Beilstein/REAXYS Number:
1098295
EC Number:
200-262-8
MDL number:
MFCD00000785
UNSPSC Code:
12000000
PubChem Substance ID:
24872015

grade

analytical standard

vapor density

5.32 (vs air)

vapor pressure

143 mmHg ( 30 °C)
91 mmHg ( 20 °C)

packaging

ampule of 1000 mg

availability

not available in Japan

technique(s)

HPLC: suitable
gas chromatography (GC): suitable

refractive index

n20/D 1.460 (lit.)

bp

76-77 °C (lit.)

mp

−23 °C (lit.)

density

1.594 g/mL at 25 °C (lit.)

application(s)

environmental

format

neat

SMILES string

ClC(Cl)(Cl)Cl

InChI

1S/CCl4/c2-1(3,4)5

InChI key

VZGDMQKNWNREIO-UHFFFAOYSA-N

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Application

Refer to the product′s Certificate of Analysis for more information on a suitable instrument technique. Contact Technical Service for further support.

pictograms

Skull and crossbonesHealth hazard
GHS06,GHS08

signalword

Danger

Hazard Classifications

Acute Tox. 3 Dermal - Acute Tox. 3 Inhalation - Acute Tox. 3 Oral - Aquatic Chronic 3 - Carc. 2 - Ozone 1 - Skin Sens. 1B - STOT RE 1 Inhalation

target_organs

Liver,Kidney

Storage Class

6.1A - Combustible acute toxic Cat. 1 and 2 / very toxic hazardous materials

wgk_germany

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

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Certificates of Analysis (COA)

Lot/Batch Number
LB91921V
LB81636V
LB77812
LB74832
LB72472

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Samar Basu
Toxicology, 189(1-2), 113-127 (2003-06-25)
Hepatic injury through carbon tetrachloride (CCl(4)) induced lipid peroxidation is well known and has been extensively used in the experimental models to understand the cellular mechanisms behind oxidative damage and further to evaluate the therapeutic potential of drugs and dietary
Lutz W D Weber et al.
Critical reviews in toxicology, 33(2), 105-136 (2003-04-24)
The use of many halogenated alkanes such as carbon tetrachloride (CCl4), chloroform (CHCl3) or iodoform (CHI3), has been banned or severely restricted because of their distinct toxicity. Yet CCl4 continues to provide an important service today as a model substance
Samuele De Minicis et al.
Hepatology (Baltimore, Md.), 59(5), 1738-1749 (2013-08-21)
Nonalcoholic fatty liver disease (NAFLD) may lead to hepatic fibrosis. Dietary habits affect gut microbiota composition, whereas endotoxins produced by Gram-negative bacteria stimulate hepatic fibrogenesis. However, the mechanisms of action and the potential effect of microbiota in the liver are
Inge Mannaerts et al.
PloS one, 8(1), e55786-e55786 (2013-02-06)
The conversion of a quiescent vitamin A storing hepatic stellate cell (HSC) to a matrix producing, contractile myofibroblast-like activated HSC is a key event in the onset of liver disease following injury of any aetiology. Previous studies have shown that
Marc Mejias et al.
Gut, 64(4), 657-666 (2014-05-23)
Antiangiogenic strategies have been proposed as a promising new approach for the therapy of portal hypertension and chronic liver disease. Pigment epithelium-derived factor (PEDF) is a powerful endogenous angiogenesis inhibitor whose role in portal hypertension remains unknown. Therefore, we aimed
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