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P9638

Sigma-Aldrich

L-α-Phosphatidylinositol 4-monophosphate sodium salt

≥98%

Synonym(s):

1,2-Diacyl-sn-glycero-3-phospho-(1-D-myo-inositol 4-phosphate) sodium-potassium salt, Diphosphoinositide sodium salt, PIP, PtdInsP

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About This Item

UNSPSC Code:
12352207

assay

≥98%

form

powder

solubility

H2O: soluble

shipped in

dry ice

storage temp.

−20°C

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Biochem/physiol Actions

Phosphorylated phospholipid membrane component, precursor of inositol phosphates. Formed in brain membranes from phosphatidylinositol by phosphatidylinositol 4-kinase.

Features and Benefits

This compound is featured on the Phosphoinositide Kinases page of the Handbook of Receptor Classification and Signal Transduction. To browse other handbook pages, click here.

Storage Class

11 - Combustible Solids

wgk_germany

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

ppe

Eyeshields, Gloves, type N95 (US)

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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C A O'Brian et al.
FEBS letters, 214(2), 339-342 (1987-04-20)
Protein kinase C(PKC) is a Ca2+- and phospholipid-dependent protein kinase which can be activated by diacylglycerol, a product of polyphosphoinositide hydrolysis. In this report, we show that the polyphosphoinositides L-alpha-phosphatidylinositol 4-monophosphate (PI 4P) and L-alpha-phosphatidylinositol 4,5-diphosphate (PI 4.5DP) can serve
L Raptis et al.
Journal of virology, 63(2), 753-758 (1989-02-01)
Polyomavirus middle tumor antigen (mT) was expressed in a line of mouse NIH 3T3 cells under control of the dexamethasone-regulatable mouse mammary tumor virus promotor. Contrary to rat F111 cells which were rendered anchorage independent by mT expression alone (L.
Siavash Fazel Darbandi et al.
Cell reports, 31(2), 107495-107495 (2020-04-16)
Tbr1 is a high-confidence autism spectrum disorder (ASD) gene encoding a transcription factor with distinct pre- and postnatal functions. Postnatally, Tbr1 conditional knockout (CKO) mutants and constitutive heterozygotes have immature dendritic spines and reduced synaptic density. Tbr1 regulates expression of

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