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H1138

Sigma-Aldrich

Horse Serum

Donor Herd, USA origin, Heat inactivated, sterile-filtered, suitable for cell culture

Synonym(s):

HS, equine sera, equine serum, horse sera, sera, serum

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About This Item

MDL number:
MFCD00164115
UNSPSC Code:
12352207
NACRES:
NA.71

biological source

horse serum

sterility

sterile-filtered

composition

Hemoglobin, ≤20 mg/dL

origin

USA origin

technique(s)

cell culture | mammalian: suitable

impurities

≤10 EU/mL Endotoxin

shipped in

dry ice

storage temp.

−20°C

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Application

Horse Serum has been used:
  • as a media supplement in the differentiation medium to culture the pheochromocytoma (PC12) cells
  • in immunocytochemistry and immunohistichemistry
  • to study the therapeutic effect of serum treatment on cardiomyocyte viability

Preparation Note

Collected from a controlled herd.
Prepared by heating at 56 °C for 30 minutes.

Storage Class

10 - Combustible liquids

wgk_germany

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

ppe

Eyeshields, Gloves

Certificates of Analysis (COA)

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Joerg Jores et al.
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Nathan J Marchant et al.
The Journal of neuroscience : the official journal of the Society for Neuroscience, 34(22), 7447-7457 (2014-05-30)
In human alcoholics, abstinence is often self-imposed, despite alcohol availability, because of the negative consequences of excessive use. During abstinence, relapse is often triggered by exposure to contexts associated with alcohol use. We recently developed a rat model that captures
Julien Dufort-Gervais et al.
Scientific reports, 10(1), 6956-6956 (2020-04-26)
Synapse loss occurs early and correlates with cognitive decline in Alzheimer's disease (AD). Synaptotoxicity is driven, at least in part, by amyloid-beta oligomers (Aβo), but the exact synaptic components targeted by Aβo remain to be identified. We here tested the
Gianluca Fulgenzi et al.
Nature communications, 11(1), 1950-1950 (2020-04-25)
BDNF signaling in hypothalamic circuitries regulates mammalian food intake. However, whether BDNF exerts metabolic effects on peripheral organs is currently unknown. Here, we show that the BDNF receptor TrkB.T1 is expressed by pancreatic β-cells where it regulates insulin release. Mice
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