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CLS3396

Corning® HTS Transwell®-24 well permeable supports

HTS Transwell-24 units w/ 0.4 μm pore polycarbonate membrane and 6.5 mm inserts, TC-treated, sterile, 2/cs

Synonym(s):

corning transwell, transwell corning

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About This Item

UNSPSC Code:
41121800
NACRES:
NB.13

material

clear polystyrene plate
flat bottom wells clear
polycarbonate membrane
round wells

description

2 HTS Transwell-24 units loaded into two open reservoir trays (catalog number CLS3395) and two individually wrapped 24 well plates

sterility

sterile

feature

lid
skirt
 plate format: 24 wells

packaging

pack of 1
case of 2

manufacturer/tradename

Corning 3396

insert working volume

0.1 mL

membrane thickness

10 μm

size

24 wells

surface area

0.33 cm2 , cell growth area

well working volume

0.6 mL

color

clear

pore size

0.4 μm pore size

suitability

suitable for (cell culture applications; optimal for cell attachment)

binding type

Tissue Culture (TC)-treated surface

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General description

Corning® HTS Transwell® 24 Well Permeable Supports

Features and Benefits:

  • Treated for optimal cell attachment
  • 0.4 μm to 3 μm pore polycarbonate or polyester membrane
  • Reservoirs may be purchased separately
  • Sterilized by gamma radiation

Legal Information

Corning is a registered trademark of Corning, Inc.
Transwell is a registered trademark of Corning, Inc.

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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Xiao-Yu Chen et al.
International journal of immunopathology and pharmacology, 32, 2058738418790318-2058738418790318 (2018-07-27)
Targeting of the programmed cell-death 1 ligand 1 (PD-L1) signal pathway is a promising treatment strategy in several cancers. The purpose of this study was to evaluate the clinical significance of PD-L1 in patients with colon adenocarcinoma (COAD). A total
Kenneth C P Cheung et al.
Nature communications, 11(1), 3595-3595 (2020-07-19)
Endothelial barrier (EB) breaching is a frequent event during inflammation, and it is followed by the rapid recovery of microvascular integrity. The molecular mechanisms of EB recovery are poorly understood. Triggering of MHC molecules by migrating T-cells is a minimal

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