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109886

Sigma-Aldrich

2-Methoxyethyl acetate

reagent grade, 98%

Synonym(s):

1-Acetoxy-2-methoxyethane, Ethylene glycol monomethyl ether acetate, Methyl Cellosolve® acetate

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About This Item

Linear Formula:
CH3COOCH2CH2OCH3
CAS Number:
110-49-6
Molecular Weight:
118.13
Beilstein/REAXYS Number:
1700761
EC Number:
203-772-9
MDL number:
MFCD00008719
UNSPSC Code:
12352108
PubChem Substance ID:
329749264
NACRES:
NA.21

grade

reagent grade

Quality Level

100

assay

98%

form

liquid

expl. lim.

0.34-6.3 %

refractive index

n20/D 1.402 (lit.)

bp

145 °C (lit.)

mp

−65 °C (lit.)

density

1.009 g/mL at 25 °C (lit.)

SMILES string

COCCOC(C)=O

InChI

1S/C5H10O3/c1-5(6)8-4-3-7-2/h3-4H2,1-2H3

InChI key

XLLIQLLCWZCATF-UHFFFAOYSA-N

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Application

2-Methoxyethyl acetate may be employed as a monomer model to evaluate the hydration structure of poly(2-methoxyethyl acrylate).

Legal Information

Cellosolve is a registered trademark of Union Carbide

signalword

Danger

Hazard Classifications

Acute Tox. 4 Dermal - Acute Tox. 4 Inhalation - Acute Tox. 4 Oral - Flam. Liq. 3 - Repr. 1B

Storage Class

3 - Flammable liquids

wgk_germany

WGK 1

flash_point_f

114.8 °F - closed cup

flash_point_c

46 °C - closed cup

Certificates of Analysis (COA)

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A new diesel oxygenate additive and its effects on engine combustion and emissions.
Yanfeng G, et al.
Applied Thermal Engineering, 27(1), 202-207 (2007)
Freitag W and Stoye D
Paints, Coatings and Solvents, (2) (2008)
Synthesis of 2-Methoxyethyl Acetate by Transesterification on the Solid Base K2CO3/Al2O3 [J].
Gong GZ, et al.
Fine Chemicals / ????, 10, 014-014 (2008)
[Hygienic regulation of ethylene glycol monomethacrylic ether in reservoir water].
I I Datsenko et al.
Gigiena i sanitariia, (9)(9), 75-76 (1989-09-01)
R J Smialowicz et al.
Fundamental and applied toxicology : official journal of the Society of Toxicology, 18(4), 621-627 (1992-05-01)
Oral dosing of adult male F344 rats with the glycol ether 2-methoxyethanol (ME) or its principal metabolite 2-methoxyacetic acid (MAA) results in the suppression of the primary plaque-forming cell (PFC) response to trinitrophenyl-lipopolysaccharide (TNP-LPS). In the present study, the PFC
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