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W244601

Sigma-Aldrich

Ethyl nitrite solution

10-20 wt. % in ethanol

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About This Item

Linear Formula:
C2H5ONO
CAS Number:
109-95-5
Molecular Weight:
75.07
MDL number:
MFCD00010257
Flavis number:
16.017

biological source

synthetic

reg. compliance

FDA 21 CFR 172.515

concentration

10-20 wt. % in ethanol

refractive index

n20/D 1.360

density

0.792 g/mL at 25 °C

organoleptic

nearly

SMILES string

CCON=O

InChI

1S/C2H5NO2/c1-2-5-3-4/h2H2,1H3

InChI key

QQZWEECEMNQSTG-UHFFFAOYSA-N

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pictograms

FlameExclamation mark
GHS02,GHS07

signalword

Danger

hcodes

H225,H319

Hazard Classifications

Eye Irrit. 2 - Flam. Liq. 2

Storage Class

3 - Flammable liquids

wgk_germany

WGK 1

flash_point_f

59.0 °F - closed cup

flash_point_c

15 °C - closed cup

ppe

Eyeshields, Faceshields, Gloves, type ABEK (EN14387) respirator filter

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Certificates of Analysis (COA)

Lot/Batch Number
60696EJ
14405JE

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J C MacDonald et al.
Acta chemica Scandinavica. Series B: Organic chemistry and biochemistry, 35(7), 485-487 (1981-01-01)
Gas chromatography--mass spectrometry demonstrates the presence of ethyl nitrite in the breath of volunteers subjects who smoke and drink concurrently. The mutagenic compound is quantitated at the ppb level by gas chromatography with the selective electron capture detector. The compound
Alessio Baccarani et al.
Journal of reconstructive microsurgery, 23(5), 257-262 (2007-09-19)
Vasospasm leading to thrombosis is a major cause of free flap failure. Flap blood flow may improve by application of vasodilating agents to the pedicle. The purpose of this study is to evaluate the effect of a novel S-nitrosylating agent
Martin P Moya et al.
Lancet (London, England), 360(9327), 141-143 (2002-07-20)
Inhaled nitric oxide is used to alleviate pulmonary hypertension and hypoxaemia, but generates toxic free radicals and oxides of nitrogen (NO(x)), which can cause rebound-hypoxia and additional pulmonary and other morbidity. To address these problems, we assessed the efficacy of
Basil M Yurcisin et al.
Annals of surgery, 257(5), 971-977 (2013-01-31)
To determine if reduction in nitric oxide bioactivity contributes to the physiological instability that occurs after brain death and, if so, to also determine in this setting whether administration of a renitrosylating agent could improve systemic physiological status. Organ function
Richard L Auten et al.
American journal of respiratory and critical care medicine, 176(3), 291-299 (2007-05-05)
Inhaled nitric oxide (NO) has been used to prevent bronchopulmonary dysplasia, but with variable results. Ethyl nitrite (ENO) forms S-nitrosothiols more readily than does NO, and resists higher-order nitrogen oxide formation. Because S-nitrosylation is a key pathway mediating many NO
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