227293
Cyclohexanecarboxamide
97%
Synonym(s):
Hexahydrobenzamide
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About This Item
Linear Formula:
C6H11CONH2
CAS Number:
Molecular Weight:
127.18
EC Number:
MDL number:
UNSPSC Code:
12352100
PubChem Substance ID:
NACRES:
NA.22
Recommended Products
assay
97%
mp
186-188 °C (lit.)
functional group
amide
SMILES string
NC(=O)C1CCCCC1
InChI
1S/C7H13NO/c8-7(9)6-4-2-1-3-5-6/h6H,1-5H2,(H2,8,9)
InChI key
PNZXMIKHJXIPEK-UHFFFAOYSA-N
General description
The enthalpy of dilution of cyclohexanecarboxamide in N,N-dimethylformamide was studied to evaluate the enthalpic interaction coefficient.
signalword
Warning
hcodes
Hazard Classifications
Eye Irrit. 2 - Skin Irrit. 2 - STOT SE 3
target_organs
Respiratory system
Storage Class
11 - Combustible Solids
wgk_germany
WGK 3
flash_point_f
Not applicable
flash_point_c
Not applicable
ppe
dust mask type N95 (US), Eyeshields, Gloves
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Dai Han et al.
Journal of integrative neuroscience, 18(3), 245-251 (2019-10-12)
The cognitive impairment associated with schizophrenia is highly prevalent and affects the overall functioning of subjects. The stimulation of the serotonin 1A receptor is a primary characteristic of some atypical antipsychotic drugs. We measured the levels of cognitive impairment using
Qing Zhu et al.
Scientific reports, 6, 19266-19266 (2016-01-15)
Vitexin, a C-glycosylated flavone present in several medicinal herbs, has showed various pharmacological activities including antinociception. The present study investigated the antinociceptive effects of vitexin in a mouse model of postoperative pain. This model was prepared by making a surgical
Enthalpies of dilution of benzamide, cyclohexanecarboxamide, and N-acetyl-N′-methyl-l-cyclohexylalaninamide dissolved in N,N-dimethylformamide at 298.15 K.
The Journal of Chemical Thermodynamics, 21(12), 1301-1306 (1989)
Marine Chazalon et al.
Neuropharmacology, 138, 315-330 (2018-06-17)
Serotonin (5-HT) neurons are involved in wake promotion and exert a strong inhibitory influence on rapid eye movement (REM) sleep. Such effects have been ascribed, at least in part to the action of 5-HT at post-synaptic 5-HT1A receptors (5-HT1AR) in
Svetlana I Sofronova et al.
The Journal of endocrinology, 235(2), 137-151 (2017-08-11)
The mechanisms of vascular alterations resulting from early thyroid hormones deficiency are poorly understood. We tested the hypothesis that antenatal/early postnatal hypothyroidism would alter the activity of endothelial NO pathway and Rho-kinase pathway, which are specific for developing vasculature. Dams
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