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SAB5600232

Sigma-Aldrich

Anti-Acetyl-Histone H3 (Lys9) antibody, Rabbit monoclonal

recombinant, expressed in HEK 293 cells, clone RM161, purified immunoglobulin

同義詞:

H3K9ac, Histone H3 (acetyl K9)

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About This Item

分類程式碼代碼:
12352203
NACRES:
NA.41

重組細胞

expressed in HEK 293 cells

抗體表格

purified immunoglobulin

抗體產品種類

primary antibodies

無性繁殖

RM161, monoclonal
recombinant monoclonal

形狀

buffered aqueous glycerol solution

物種活性

vertebrates

技術

ChIP: 2-10 μg/mL
ELISA: 0.5-2 μg/mL
immunocytochemistry: 0.5-2 μg/mL
immunohistochemistry: 0.1—1 μg/mL
multiplexing: 0.05-0.5 μg/mL
western blot: 0.25— 1 μg/mL

同型

IgG

NCBI登錄號

UniProt登錄號

運輸包裝

wet ice

儲存溫度

−20°C

目標翻譯後修改

acetylation (Lys9)

基因資訊

human ... H3C1(8350)

特異性

This antibody reacts to Histone H3 acetylated at Lysine 9 (K9ac). No cross reactivity with other acetylated Lysines in histone H3.

免疫原

An acetyl-peptide corresponding to the Acetyl-Histone H3 (Lys9)

外觀

Solution in phosphate buffered saline containing 50% Glycerol/PBS with 1% BSA and 0.09% sodium azide.

免責聲明

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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儲存類別代碼

10 - Combustible liquids

水污染物質分類(WGK)

WGK 2

閃點(°F)

Not applicable

閃點(°C)

Not applicable


分析證明 (COA)

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Lin Xiao et al.
Clinical epigenetics, 13(1), 53-53 (2021-03-12)
Intervertebral disc degeneration (IDD) is a major cause of lower back pain. This study aimed at exploring the effects of histone deacetylase 4 (HDAC4) and its upstream and downstream signaling molecules on IDD development. A murine IDD model was established
Hang Xue et al.
American journal of physiology. Cell physiology, 319(2), C381-C391 (2020-06-04)
Several microRNAs (miRNAs or miRs) regulate cerebral ischemic injury outcomes; however, little is known about the role of miR-539-5p during cerebral ischemic injury or the postischemic state. Cerebral ischemic injury was modeled in vitro by exposing human cortical neurons to

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