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EXTRA2

Sigma-Aldrich

小鼠ExtrAvidin®过氧化物酶染色试剂盒 山羊抗

同義詞:

ExtrAvidin®过氧化物酶染色试剂盒

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About This Item

分類程式碼代碼:
12352203
NACRES:
NA.32

抗體產品種類

secondary antibodies

用途

sufficient for 2,000 tests ELISA, Immunoblotting
sufficient for 200 tests Immunohistology

運輸包裝

wet ice

儲存溫度

2-8°C

目標翻譯後修改

unmodified

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一般說明

包含可与一抗联用的通用试剂,适用于免疫组化、ELISA和免疫印迹。
ExtrAvidin®是一种形式独特的亲和素,由Sigma独家供货,可将亲和素对生物素的高特异性和高亲和性与在生理条件下的低非特异性有机结合在一起。ExtrAvidin®过氧化物酶具有灵敏度高、本底水平低的特点。

應用

Applications in which this antibody has been used successfully, and the associated peer-reviewed papers, are given below.
Immunohistochemistry (1 paper)

特點和優勢

  • 可用于免疫组化、ELISA和免疫印迹检测。
  • 使用human IgG和IgM对亲和纯化的抗体进行吸附,可最大限度减少交叉反应。
  • 生物素标记的抗体含有一段间隔肽链,可提高其与ExtrAvidin® 偶联物的亲和性。

成分

• 3 mL ExtrAvidin®-过氧化物酶
• 3 mL生物素标记的二抗
• 附带完整使用说明和实验方案

法律資訊

ExtrAvidin is a registered trademark of Merck KGaA, Darmstadt, Germany

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儲存類別代碼

12 - Non Combustible Liquids

水污染物質分類(WGK)

WGK 3

閃點(°F)

Not applicable

閃點(°C)

Not applicable

個人防護裝備

Eyeshields, Gloves, multi-purpose combination respirator cartridge (US)


分析證明 (COA)

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存取文件庫

Matheus L Medeiros et al.
Journal of inflammation research, 14, 6477-6489 (2021-12-10)
Methylglyoxal (MGO) is a highly reactive dicarbonyl species implicated in diabetic-associated diseases. Acute lung injury (ALI) symptoms and prognosis are worsened by diabetes and obesity. Here, we hypothesized that elevated MGO levels aggravate ALI, which can be prevented by metformin.
Akila L Oliveira et al.
Frontiers in physiology, 13, 860342-860342 (2022-04-15)
Methylglyoxal (MGO) is a highly reactive dicarbonyl compound implicated in diabetes-associated diseases. In vascular tissues, MGO induces the formation of advanced glycation end products (AGEs) that bounds its receptor RAGE, initiating the downstream tissue injury. Outside the cardiovascular system, MGO
Vladimirs Pilipenko et al.
Neuropharmacology, 144, 319-326 (2018-11-09)
Early manifestations of Alzheimer's disease (AD) include neuroinflammation, disrupted neurotransmission and cognitive deficits. Impairment of the GABAergic system is essentially involved in the pathogenesis of AD. Traditionally, agonists of GABAA receptors at doses above 1 mg/kg are known to possess memory

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