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Merck
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重要文件

D0513

Supelco

狄布卡因

analytical standard

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About This Item

經驗公式(希爾表示法):
C20H29N3O2
CAS號碼:
分子量::
343.46
EC號碼:
MDL號碼:
分類程式碼代碼:
41116107
PubChem物質ID:

等級

analytical standard

技術

HPLC: suitable
gas chromatography (GC): suitable

應用

cleaning products
cosmetics
food and beverages
forensics and toxicology
personal care
pharmaceutical (small molecule)
veterinary

格式

neat

SMILES 字串

CCCCOc1cc(C(=O)NCCN(CC)CC)c2ccccc2n1

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應用

Refer to the product′s Certificate of Analysis for more information on a suitable instrument technique. Contact Technical Service for further support.

生化/生理作用

强效、长效局部麻醉剂

象形圖

Environment

危險聲明

防範說明

危險分類

Aquatic Chronic 2

儲存類別代碼

11 - Combustible Solids

水污染物質分類(WGK)

WGK 3

閃點(°F)

Not applicable

閃點(°C)

Not applicable

個人防護裝備

dust mask type N95 (US), Eyeshields, Faceshields, Gloves


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分析證明 (COA)

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Bryan Heit et al.
American journal of physiology. Cell physiology, 300(1), C33-C41 (2010-10-12)
Electrostatic interactions with negative lipids contribute to the subcellular localization of polycationic proteins. In situ measurements using cytosolic probes of surface charge indicate that normal mitochondria are not noticeably electronegative. However, during apoptosis mitochondria accrue negative charge and acquire the
Ana Peropadre et al.
Chemical research in toxicology, 24(2), 185-192 (2011-01-26)
A wide range of cationic amphiphilic drugs (CADs) from different therapeutic areas are known to cause phospholipidosis both in vivo and in vitro. Although the relevance of this storage disorder for human health remains uncertain, CADs have been repeatedly associated
V Arizza et al.
Fish & shellfish immunology, 30(4-5), 1014-1023 (2011-02-04)
Hemocytes from the ascidian Ciona intestinalis exert in vitro Ca²+-dependent cytotoxic activity toward mammalian erythrocytes and K562 cells. To examine the lytic mechanism, hemocyte populations were separated (B1-B6 bands) through a Percoll discontinuous density gradient, the hemocyte cytotoxic activity (HCA)
W Christopher Risher et al.
PloS one, 6(7), e22351-e22351 (2011-07-27)
Spreading depolarizations that occur in patients with malignant stroke, subarachnoid/intracranial hemorrhage, and traumatic brain injury are known to facilitate neuronal damage in metabolically compromised brain tissue. The dramatic failure of brain ion homeostasis caused by propagating spreading depolarizations results in
Heather A Douglas et al.
Journal of neurophysiology, 105(4), 1482-1494 (2011-01-29)
Recurring waves of peri-infarct depolarizations (PIDs) propagate across gray matter in the hours and days following stroke, expanding the primary site of injury. Ischemic depolarization (termed anoxic depolarization or AD in live brain slices) is PID-like but immediately arises in

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