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MAB1570

Sigma-Aldrich

Anti-NMDAR1 Antibody, CT, C2 cassette, clone 2C6.2

clone 2C6.2, Chemicon®, from mouse

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About This Item

分類程式碼代碼:
12352203
eCl@ss:
32160702
NACRES:
NA.41

生物源

mouse

抗體表格

purified immunoglobulin

抗體產品種類

primary antibodies

無性繁殖

2C6.2, monoclonal

物種活性

rat

製造商/商標名

Chemicon®

技術

immunohistochemistry: suitable
western blot: suitable

同型

IgG2b

UniProt登錄號

運輸包裝

wet ice

目標翻譯後修改

unmodified

基因資訊

特異性

NMDAR1, C-terminal, C2 cassette

免疫原

Epitope: C-terminus, C2 cassette
Synthetic peptide (LQNQKDTVLPRRAIEREEGQLQLCSRHRES) corresponding to the C-terminus of rat NMDA receptor subunit.

應用

Anti-NMDAR1 Antibody, C-terminus, C2 cassette, clone 2C6.2 is an antibody against NMDAR1 for use in IH & WB.
Research Category
Neuroscience
Research Sub Category
Neurotransmitters & Receptors
Western blot: 1:100-1:500

Immunohistochemistry: 1:100-1:500 using paraformaldehyde or paraformaldehyde/glutaraldehyde fixed tissue with light and electron microscopy.

Optimal working dilutions must be determined by the end user.

外觀

Format: Purified
Purified immunoglobulin. Liquid in 0.02M Phosphate buffer, 0.25 M NaCl with 0.1% sodium azide.

儲存和穩定性

Maintain at 2-8°C in undiluted aliquots for up to 6 months.

其他說明

Concentration: Please refer to the Certificate of Analysis for the lot-specific concentration.

法律資訊

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

免責聲明

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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儲存類別代碼

10 - Combustible liquids

水污染物質分類(WGK)

WGK 2

閃點(°F)

Not applicable

閃點(°C)

Not applicable


分析證明 (COA)

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Tom J Phillips et al.
Scientific reports, 7(1), 9079-9079 (2017-08-24)
Some neuropsychiatric disease, including schizophrenia, may originate during prenatal development, following periods of gestational hypoxia and placental oxidative stress. Here we investigated if gestational hypoxia promotes damaging secretions from the placenta that affect fetal development and whether a mitochondria-targeted antioxidant

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