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Merck
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重要文件

AB5725

Sigma-Aldrich

Anti-Gephyrin Antibody

Chemicon®, from rabbit

同義詞:

Anti-GEPH, Anti-GPH, Anti-GPHRYN, Anti-HKPX1, Anti-MOCODC

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About This Item

分類程式碼代碼:
12352203
eCl@ss:
32160702
NACRES:
NA.41

生物源

rabbit

抗體表格

affinity purified immunoglobulin

抗體產品種類

primary antibodies

無性繁殖

polyclonal

純化經由

affinity chromatography

物種活性

mouse, rat

製造商/商標名

Chemicon®

技術

western blot: suitable

UniProt登錄號

運輸包裝

wet ice

目標翻譯後修改

unmodified

基因資訊

human ... GPHN(10243)

特異性

Gephyrin

免疫原

Synthetic peptide from rat gephyrin.

應用

Anti-Gephyrin Antibody is an antibody against Gephyrin for use in WB.
Research Category
Neuroscience
Research Sub Category
Synapse & Synaptic Biology
Western blot: 0.5-1.0 μg/mL using ECL on mouse cerebellum tissue lysate. The antibody reacts with a protein of ~93 kDa. An additional band of ~35 kDa may also be seen depending on sample used.

Optimal working dilutions must be determined by the end user.

外觀

Affinity purified immunoglobulin. Liquid.

儲存和穩定性

Maintain at +2-8°C in undiluted aliquots for up to 6 months after date of receipt.

其他說明

Concentration: Please refer to the Certificate of Analysis for the lot-specific concentration.

法律資訊

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

免責聲明

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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儲存類別代碼

10 - Combustible liquids

水污染物質分類(WGK)

WGK 2

閃點(°F)

Not applicable

閃點(°C)

Not applicable


分析證明 (COA)

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Dibyadeep Datta et al.
Journal of neuroinflammation, 17(1), 8-8 (2020-01-08)
Cognitive impairment in schizophrenia, aging, and Alzheimer's disease is associated with spine and synapse loss from the dorsolateral prefrontal cortex (dlPFC) layer III. Complement cascade signaling is critical in driving spine loss and disease pathogenesis. Complement signaling is initiated by
Yvonne M Ulrich-Lai et al.
Brain structure & function, 221(6), 3141-3156 (2015-08-08)
A history of eating highly palatable foods reduces physiological and emotional responses to stress. For instance, we have previously shown that limited sucrose intake (4 ml of 30 % sucrose twice daily for 14 days) reduces hypothalamic-pituitary-adrenocortical (HPA) axis responses to stress. However
Wen Zhou et al.
International journal of molecular medicine, 38(5), 1481-1489 (2016-12-28)
The medial vestibular nucleus (MVN) and the cerebellar flocculus have been known to be the key areas involved in vestibular compensation (VC) following unilateral labyrinthectomy (UL). In this study, we examined the role of gephyrin and glycine receptor (GlyR) in VC using Sprague-Dawley rats
Jessica S Nikitczuk et al.
Hippocampus, 24(8), 943-962 (2014-04-23)
N-Cadherin and β-catenin form a transsynaptic adhesion complex required for spine and synapse development. In adulthood, N-cadherin mediates persistent synaptic plasticity, but whether the role of N-cadherin at mature synapses is similar to that at developing synapses is unclear. To

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