182517
Arp2/3复合物抑制剂I，失活对照，CK-689
The Arp2/3 Complex Inhibitor I, Inactive Control, CK-689, also referenced under CAS 170930-46-8, controls the biological activity of Arp2/3. This small molecule/inhibitor is primarily used for Cell Structure applications.
同義詞:
Arp2/3复合物抑制剂I，失活对照，CK-689
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About This Item
推薦產品
品質等級
化驗
≥95% (HPLC)
形狀
solid
製造商／商標名
Calbiochem®
儲存條件
OK to freeze
protect from light
顏色
off-white
溶解度
DMSO: 50 mg/mL
運輸包裝
ambient
儲存溫度
2-8°C
一般說明
在基于细胞和无细胞的实验中，细胞可渗透的吲哚基-甲氧基乙酰胺均不显示Arp2/3抑制活性，并且可作为CK-666（目录编号182515）的无活性对照。
在基于细胞和无细胞的实验中，细胞可渗透的吲哚基-甲氧基乙酰胺均不显示Arp2/3抑制活性，并且可作为CK-666（目录编号182515）的无活性对照。
包裝
用惰性气体包装
警告
毒性：标准处理（A）
重構
复溶后，等分并冷冻保存（-20°C）。储备液在-20°C条件下可稳定保存6个月。
其他說明
Nolen, B.J., et al. 2009.Nature460, 1031.
法律資訊
CALBIOCHEM is a registered trademark of Merck KGaA, Darmstadt, Germany
儲存類別代碼
11 - Combustible Solids
水污染物質分類（WGK）
WGK 3
閃點（°F）
Not applicable
閃點（°C）
Not applicable
分析證明 (COA)
輸入產品批次/批號來搜索 分析證明 (COA)。在產品’s標籤上找到批次和批號，寫有 ‘Lot’或‘Batch’.。
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Antigen-induced B-cell receptor (BCR) signaling is critical for initiating and regulating B-cell activation. The actin cytoskeleton plays essential roles in BCR signaling. Upon encountering cell-surface antigens, actin-driven B-cell spreading amplifies signaling, while B-cell contraction following spreading leads to signal attenuation.
Giulia Cazzagon et al.
iScience, 26(7), 107129-107129 (2023-07-12)
Although the formin-nucleated actomyosin cortex has been shown to drive the changes in cell shape that accompany animal cell division in both symmetric and asymmetric cell divisions, the mitotic role of cortical Arp2/3-nucleated actin networks remain unclear. Here using asymmetrically
Florian Gaertner et al.
Developmental cell, 57(1), 47-62 (2021-12-18)
When crawling through the body, leukocytes often traverse tissues that are densely packed with extracellular matrix and other cells, and this raises the question: How do leukocytes overcome compressive mechanical loads? Here, we show that the actin cortex of leukocytes
An ancestral mycobacterial effector promotes dissemination of infection.
Saelens, et al.
Cell, 185, 4507-4525 (2023)
Domokos I Lauko et al.
Molecular biology of the cell, 32(16), 1433-1445 (2021-06-17)
The baculovirus Autographa californica multiple nucleopolyhedrovirus (AcMNPV), a pathogen of lepidopteran insects, has a striking dependence on the host cell actin cytoskeleton. During the delayed-early stage of infection, AcMNPV was shown to induce the accumulation of actin at the cortex of infected cells. However, the dynamics and
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