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Merck
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SAB1305952

Sigma-Aldrich

ANTI-TIAM1(C-TERMINAL) antibody produced in rabbit

affinity isolated antibody, buffered aqueous solution

别名:

T-lymphoma invasion and metastasis-inducing protein 1, TIAM1

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About This Item

UNSPSC代码:
12352203
NACRES:
NA.41

生物来源

rabbit

抗体形式

affinity isolated antibody

抗体产品类型

primary antibodies

克隆

polyclonal

表单

buffered aqueous solution

分子量

177508 Da

种属反应性

human

技术

western blot: 1:1000

NCBI登记号

UniProt登记号

运输

wet ice

储存温度

−20°C

靶向翻译后修饰

unmodified

基因信息

human ... TIAM1(7074)

外形

Supplied in PBS with 0.09% (W/V) sodium azide

免责声明

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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储存分类代码

12 - Non Combustible Liquids

闪点(°F)

Not applicable

闪点(°C)

Not applicable


历史批次信息供参考:

分析证书(COA)

Lot/Batch Number

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Daisuke Izumi et al.
Cell death & disease, 10(4), 267-267 (2019-03-21)
Accumulating evidence suggests that cancer cells with stem cell-like features have higher resistance to chemotherapeutic agents. Herein, we identified T-lymphoma invasion and metastasis-inducing protein-1 (TIAM1) as one of the Wnt-signaling associated genes which drives self-renewal and its expression is upregulated
Xiaoping Zhang et al.
Oncogene, 38(5), 699-715 (2018-09-02)
Anaplastic thyroid cancer (ATC) is associated with poor prognosis and is often untreatable. MicroRNA 483-3p (miR-483) and partitioning-defective 3 (Pard3), a member of the Pard family, have functions and regulatory mechanisms in ATC. The abnormal regulation of miR-483 may play
Hidetsugu Kawai et al.
Neoplasia (New York, N.Y.), 21(8), 788-801 (2019-07-06)
Hematological malignancies harboring various ABL1 fusions are expected to be sensitive to tyrosine kinase inhibitors (TKIs), similar to those with BCR-ABL1. However, SEPT9-ABL1 exhibits TKI resistance both in vitro and in vivo. SEPT9-ABL1 has the same ABL1 region as seen
Takeo Saneyoshi et al.
Neuron, 102(6), 1199-1210 (2019-05-13)
Long-term synaptic plasticity requires a mechanism that converts short Ca2+ pulses into persistent biochemical signaling to maintain changes in the synaptic structure and function. Here, we present a novel mechanism of a positive feedback loop, formed by a reciprocally activating

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