推荐产品
形狀
solid
存貨情形
available only in Japan
bp
287 °C/265 mmHg (lit.)
mp
126-128 °C (lit.)
應用
peptide synthesis
SMILES 字串
CC1=NN(C(=O)C1)c2ccccc2
InChI
1S/C10H10N2O/c1-8-7-10(13)12(11-8)9-5-3-2-4-6-9/h2-6H,7H2,1H3
InChI 密鑰
QELUYTUMUWHWMC-UHFFFAOYSA-N
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適合性
environmental analysis
訊號詞
Warning
危險聲明
危險分類
Acute Tox. 4 Oral - Eye Irrit. 2
儲存類別代碼
11 - Combustible Solids
水污染物質分類（WGK）
WGK 1
個人防護裝備
dust mask type N95 (US), Eyeshields, Gloves
Ajmal Ahmad et al.
Molecular and cellular biochemistry, 367(1-2), 215-225 (2012-06-01)
Stroke is a life-threatening disease with major cause of mortality and morbidity worldwide. The neuronal damage following cerebral ischemia is a serious risk to stroke patients. Oxidative stress and apoptotic damage play an important role in cerebral ischemic pathogenesis and
Laura Monturiol-Gross et al.
The Journal of infectious diseases, 206(8), 1218-1226 (2012-08-21)
Clostridium perfringens, the most broadly distributed pathogen in nature, produces a prototype phospholipase C, also called α-toxin, which plays a key role in the pathogenesis of gas gangrene. α-Toxin causes plasma membrane disruption at high concentrations, but the role of
Manabu Ohta et al.
Brain research, 1490, 184-192 (2012-09-18)
Traumatic axonal injury (TAI), a feature of traumatic brain injury (TBI), progressively evolves over hours through impaired axonal transport and is thought to be a major contributor to cognitive dysfunction. In spite of various studies suggesting that pharmacologic or physiologic
Mitsugi Shimoda et al.
World journal of gastroenterology, 18(27), 3520-3526 (2012-07-25)
To investigate the effect of E3-methyl-1-phenyl-2-pyrazolin-5-one (Edr) on hepatic ischemia-reperfusion (I/R) injury and liver regeneration in a porcine hepatectomy model. One hour ischemia was induced by occluding the vessels and the bile duct of the right and median lobes. A
Nobukazu Miyamoto et al.
Stroke, 44(12), 3516-3521 (2013-09-28)
White matter injury caused by cerebral hypoperfusion may contribute to the pathophysiology of vascular dementia and stroke, but the underlying mechanisms remain to be fully defined. Here, we test the hypothesis that oxidative stress interferes with endogenous white matter repair
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