所有图片(1)
About This Item
经验公式（希尔记法）:
H2O2
CAS号:
分子量:
34.01
Beilstein:
3587191
MDL號碼:
分類程式碼代碼:
12352304
PubChem物質ID:
推荐产品
等級
JIS special grade
蒸汽壓力
23.3 mmHg ( 30 °C)
形狀
liquid
反應適用性
reagent type: oxidant
存貨情形
available only in Japan
濃度
30.0-35.5%
密度
1.13 g/cm3
SMILES 字串
OO
InChI
1S/H2O2/c1-2/h1-2H
InChI 密鑰
MHAJPDPJQMAIIY-UHFFFAOYSA-N
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訊號詞
Danger
危險分類
Acute Tox. 4 Oral - Aquatic Chronic 3 - Eye Dam. 1 - Skin Irrit. 2 - STOT SE 3
標靶器官
Respiratory system
儲存類別代碼
5.1B - Oxidizing hazardous materials
水污染物質分類（WGK）
WGK 1
閃點（°F）
Not applicable
閃點（°C）
Not applicable
Nicola J Jordan et al.
Breast cancer research : BCR, 16(1), R12-R12 (2014-01-25)
Upregulation of PI3K/Akt/mTOR signalling in endocrine-resistant breast cancer (BC) has identified mTOR as an attractive target alongside anti-hormones to control resistance. RAD001 (everolimus/Afinitor®), an allosteric mTOR inhibitor, is proving valuable in this setting; however, some patients are inherently refractory or
Serena Lunardi et al.
Oncotarget, 5(22), 11064-11080 (2014-11-22)
Pancreatic ductal adenocarcinoma (PDAC) is characterized by an abundant desmoplastic reaction driven by pancreatic stellate cells (PSCs) that contributes to tumor progression. Here we sought to characterize the interactions between pancreatic cancer cells (PCCs) and PSCs that affect the inflammatory
Danny C LeBert et al.
Development (Cambridge, England), 142(12), 2136-2146 (2015-05-28)
Acute and chronic injuries are characterized by leukocyte infiltration into tissues. Although matrix metalloproteinase 9 (Mmp9) has been implicated in both conditions, its role in wound repair remains unclear. We previously reported a zebrafish chronic inflammation mutant caused by an
X Sun et al.
Oncogene, 34(45), 5617-5625 (2015-03-03)
Ferroptosis is an iron-dependent form of non-apoptotic cell death, but its molecular mechanism remains largely unknown. Here, we demonstrate that heat shock protein beta-1 (HSPB1) is a negative regulator of ferroptotic cancer cell death. Erastin, a specific ferroptosis-inducing compound, stimulates
Laetitia Francelle et al.
Human molecular genetics, 24(6), 1563-1573 (2014-11-16)
The mechanisms underlying preferential atrophy of the striatum in Huntington's disease (HD) are unknown. One hypothesis is that a set of gene products preferentially expressed in the striatum could determine the particular vulnerability of this brain region to mutant huntingtin
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