推荐产品
形狀
liquid
存貨情形
available only in Japan
濃度
3.2 mM
SMILES 字串
Cl[Ca]Cl
InChI
1S/Ca.2ClH/h;2*1H/q+2;;/p-2
InChI 密鑰
UXVMQQNJUSDDNG-UHFFFAOYSA-L
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訊號詞
Warning
危險聲明
危險分類
Eye Irrit. 2
儲存類別代碼
10 - Combustible liquids
水污染物質分類（WGK）
WGK 1
閃點（°F）
Not applicable
閃點（°C）
Not applicable
個人防護裝備
Eyeshields, Faceshields, Gloves, type ABEK (EN14387) respirator filter
Dino Demirovic et al.
Journal of cell communication and signaling, 8(4), 333-339 (2014-09-07)
Repeated exposure to mild heat shock (HS) has been shown to induce a wide range of health promoting hormetic effects in various biological systems, including human cells undergoing aging in vitro. In order to understand how cells distinguish between mild
Kaisa Kyöstilä et al.
PLoS genetics, 11(4), e1005169-e1005169 (2015-04-16)
Inherited neurodegenerative disorders are debilitating diseases that occur across different species. We have performed clinical, pathological and genetic studies to characterize a novel canine neurodegenerative disease present in the Lagotto Romagnolo dog breed. Affected dogs suffer from progressive cerebellar ataxia
Shigeru Komaba et al.
PloS one, 10(9), e0138012-e0138012 (2015-09-12)
Amino acid transporters (AATers) in the brush border of the apical plasma membrane (APM) of renal proximal tubule (PT) cells mediate amino acid transport (AAT). We found that the membrane-associated class I myosin myosin 1b (Myo1b) localized at the apical
Konstantinos Malliaras et al.
EMBO molecular medicine, 6(6), 760-777 (2014-05-07)
Controversy surrounds the identity, origin, and physiologic role of endogenous cardiomyocyte progenitors in adult mammals. Using an inducible genetic labeling approach to identify small non-myocyte cells expressing cardiac markers, we find that activated endogenous cardioblasts are rarely evident in the
Francesca Maltecca et al.
The Journal of clinical investigation, 125(1), 263-274 (2014-12-09)
Spinocerebellar ataxia type 28 (SCA28) is a neurodegenerative disease caused by mutations of the mitochondrial protease AFG3L2. The SCA28 mouse model, which is haploinsufficient for Afg3l2, exhibits a progressive decline in motor function and displays dark degeneration of Purkinje cells
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