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56419C

SAFC

Minimum Essential Medium

with Earle′s Balanced Salts, with 2.0 mM L-glutamine, without sodium bicarbonate, dry powder, suitable for cell culture

Pharma Manufacturing

Synonym(s):

Minimum Essential Medium Eagle, MEM

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About This Item

UNSPSC Code:
12352200
NACRES:
NA.71

description

for research or for further manufacturing use

Quality Level

500

form

dry powder

technique(s)

cell culture | mammalian: suitable

components

Earle’s salts (5% CO2): yes
NaHCO3: no
L-glutamine: 292 mg/L
phenol red: 11 mg/L

shipped in

ambient

storage temp.

2-8°C

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Quantity

Formulated to contain 9.5 grams of powder per liter of medium.

Storage Class Code

11 - Combustible Solids

WGK

WGK 3

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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Hepatocellular carcinoma is one of the most fatal cancers worldwide. Propofol is an intravenous anesthetic extensively used in clinical. Herein, we tested the anticancer activity of propofol on hepatocellular carcinoma, along with the internal molecular mechanism related to lncRNA DiGeorge
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Influenza pneumonia causes high mortality every year, and pandemic episodes kill millions of people. Influenza-related mortality has been variously ascribed to an ineffective host response that fails to limit viral replication, an excessive host inflammatory response that results in lung
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Many viruses attach to target cells by binding to cell-surface glycans. To gain a better understanding of strategies used by viruses to engage carbohydrate receptors, we determined the crystal structures of reovirus attachment protein σ1 in complex with α-2,3-sialyllactose, α-2,6-sialyllactose
Wenxin Wu et al.
Virology, 396(2), 178-188 (2009-11-17)
We studied cytokine responses to influenza virus PR8 (H1N1) and Oklahoma/309/06 (OK/06, H3N2) in a novel human lung tissue model. Exposure of the model to influenza virus rapidly activated the mitogen-activated protein kinase signaling (MAPK) pathways ERK, p38 and JNK.
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