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Key Documents

P5171

Sigma-Aldrich

Endoproteinase Arg-C from mouse submaxillary gland

Type XX-S, lyophilized powder, 250-800 units/mg protein (Lowry)

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About This Item

CAS Number:
Enzyme Commission number:
EC Number:
MDL number:
UNSPSC Code:
12352204

type

Type XX-S

Quality Level

form

lyophilized powder

specific activity

250-800 units/mg protein (Lowry)

composition

Protein, 3.0-7.0%

analyte chemical class(es)

amino acids

storage temp.

2-8°C

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Unit Definition

One unit will hydrolyze 1.0 μmole of Nα-p-tosyl-L-arginine methyl ester per min at pH 8.0 at 25 °C.

Physical form

Lyophilized powder containing NaCl and sodium acetate

pictograms

Health hazardExclamation mark

signalword

Danger

Hazard Classifications

Eye Irrit. 2 - Resp. Sens. 1 - Skin Irrit. 2 - STOT SE 3

target_organs

Respiratory system

Storage Class

11 - Combustible Solids

wgk_germany

WGK 3


Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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Duncan J Campbell et al.
Clinical and experimental pharmacology & physiology, 38(9), 623-631 (2011-07-09)
1. Aliskiren is a renin inhibitor with an IC(50) of 0.6 nmol/L for human renin, 4.5 nmol/L for mouse renin and 80 nmol/L for rat renin. 2. In the present study, we compared the effects of aliskiren (10 mg/kg per day)
Rui-Yan Lu et al.
CNS neuroscience & therapeutics, 18(5), 395-399 (2012-04-27)
The aims of this study were to find out whether kallikrein could induce angiogenesis and affect the cerebral blood flow (rCBF) in the early period after cerebral ischemia/reperfusion (CI/R). The adenovirus carried human tissue kallikrein (HTK) gene was administrated into
Prostate-specific antigen kallikrein and non-ST elevation myocardial infarction.
Salvatore Patanè
International journal of cardiology, 149(3), 392-393 (2011-04-01)
Hui-Zhen Yu et al.
Molecular and cellular biochemistry, 360(1-2), 363-371 (2011-10-01)
Tissue kallikrein 1 cleaves kininogen substrate to produce vasoactive kinin peptides that have been implicated in inhibiting neointimal hyperplasia in rat carotid arteries after balloon injury. However, its effects on the proliferation, cell cycle and its mechanisms, for example, cyclin-dependent
Prostate-specific antigen kallikrein is not a marker of non-ST elevation acute coronary syndrome.
Alberto Dominguez-Rodriguez et al.
International journal of cardiology, 149(1), 141-142 (2011-03-05)

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