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Merck
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Key Documents

ST1551

Sigma-Aldrich

Anti-GGT1 Mouse mAb (1F9)

liquid, clone 1F9, Calbiochem®

Sinónimos:

Anti-γ-Glutamyltransferase1

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About This Item

UNSPSC Code:
12352203
NACRES:
NA.41

biological source

mouse

Quality Level

antibody form

purified antibody

antibody product type

primary antibodies

clone

1F9, monoclonal

form

liquid

does not contain

preservative

species reactivity

human, mouse

manufacturer/tradename

Calbiochem®

storage condition

OK to freeze
avoid repeated freeze/thaw cycles

isotype

IgG2α

shipped in

wet ice

storage temp.

−20°C

target post-translational modification

unmodified

Gene Information

human ... GGT1(2678)

General description

Anti-GGT1, mouse monoclonal, clone 1F9, recognizes the ~60 kDa GGT1 protein in NIH3T3 cells and human testes tissue. It is validated for Western blotting & immunohistochemistry on paraffin sections.
Purified mouse monoclonal antibody. Recognizes the ~60 kDa GGT1 protein.
Recognizes the ~60 kDa GGT1 protein in NIH3T3 cells and human testes tissue.

Immunogen

A recombinant polypeptide corresponding to amino acids 381-471 of human GGT1, expressed as a GST fusion protein

Warning

Toxicity: Regulatory Review (Z)

Analysis Note

Positive Control
NIH3T3 cells, Human testes tissue

Legal Information

CALBIOCHEM is a registered trademark of Merck KGaA, Darmstadt, Germany

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Storage Class

12 - Non Combustible Liquids

wgk_germany

nwg

flash_point_f

Not applicable

flash_point_c

Not applicable


Certificados de análisis (COA)

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Julieta N Caballero et al.
PloS one, 8(6), e65364-e65364 (2013-06-21)
Acquisition of fertilization ability by spermatozoa during epididymal transit occurs in part by the transfer of molecules from membranous vesicles called epididymosomes. Epididymosomes are heterogeneous in terms of both size and molecular composition. Exosomes and other related small membranous vesicles
Shenaz Khan et al.
JCI insight, 5(15) (2020-07-03)
Kidney disease is one of the most devastating complications of diabetes, and tubular atrophy predicts diabetic kidney disease (DKD) progression to end-stage renal disease. We have proposed that fatty acids bound to albumin contribute to tubular atrophy by inducing lipotoxicity

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