推荐产品
形狀
solution
儲存期限
Expiry date on the label.
IVD
for in vitro diagnostic use
濃度
0.5 % (w/v) in water
應用
hematology
histology
儲存溫度
room temp
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應用
通用细胞质复染。 与苏木精和曙红染色一起使用。
其他說明
经认证的曙红Y,水中含量为0.5%(w/v)。 未酸化。
儲存類別代碼
10 - Combustible liquids
水污染物質分類(WGK)
WGK 2
閃點(°F)
Not applicable
閃點(°C)
Not applicable
個人防護裝備
Eyeshields, Gloves, multi-purpose combination respirator cartridge (US)
其他客户在看
Journal of trace elements in medicine and biology : organ of the Society for Minerals and Trace Elements (GMS), 44, 71-75 (2017-10-03)
The influence of rhodanine and haematoxylin and eosin (HE) staining on the copper distribution and concentration in liver needle biopsy samples originating from patients with Wilson's disease (WD), a rare autosomal recessive inherited disorder of the copper metabolism, is investigated.
Cell, 182(4), 976-991 (2020-07-24)
Although complex inflammatory-like alterations are observed around the amyloid plaques of Alzheimer's disease (AD), little is known about the molecular changes and cellular interactions that characterize this response. We investigate here, in an AD mouse model, the transcriptional changes occurring
Journal of experimental & clinical cancer research : CR, 39(1), 2-2 (2020-01-09)
Quiescent/slow cycling cells have been identified in several tumors and correlated with therapy resistance. However, the features of chemoresistant populations and the molecular factors linking quiescence to chemoresistance are largely unknown. A population of chemoresistant quiescent/slow cycling cells was isolated
Nature communications, 11(1), 3360-3360 (2020-07-06)
Nonalcoholic fatty liver disease (NAFLD) is considered the next major health epidemic with an estimated 25% worldwide prevalence. No drugs have yet been approved and NAFLD remains a major unmet need. Here, we identify MCJ (Methylation-Controlled J protein) as a
Nature communications, 8, 14143-14143 (2017-02-01)
Duchenne muscular dystrophy (DMD) is characterized by muscle degeneration and progressive weakness. There is considerable inter-patient variability in disease onset and progression, which can confound the results of clinical trials. Here we show that a common null polymorphism (R577X) in
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