推荐产品
化驗
98%
形狀
liquid
折射率
n20/D 1.534 (lit.)
bp
220 °C (lit.)
mp
11-13 °C (lit.)
密度
1.102 g/mL at 25 °C (lit.)
SMILES 字串
CC(=O)c1cccnc1
InChI
1S/C7H7NO/c1-6(9)7-3-2-4-8-5-7/h2-5H,1H3
InChI 密鑰
WEGYGNROSJDEIW-UHFFFAOYSA-N
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訊號詞
Danger
危險聲明
危險分類
Acute Tox. 3 Oral - Skin Irrit. 2
儲存類別代碼
6.1C - Combustible acute toxic Cat.3 / toxic compounds or compounds which causing chronic effects
水污染物質分類（WGK）
WGK 1
閃點（°F）
219.2 °F - closed cup
閃點（°C）
104 °C - closed cup
個人防護裝備
Eyeshields, Faceshields, Gloves, type ABEK (EN14387) respirator filter
其他客户在看
Matasha Dhar et al.
Nature communications, 7, 12938-12938 (2016-09-22)
Neurodegenerative lesions induce sprouting of new collaterals from surviving axons, but the extent to which this form of axonal remodelling alters brain functional structure remains unclear. To understand how collateral sprouting proceeds in the adult brain, we imaged post-lesion sprouting
Huawen Lin et al.
PLoS genetics, 6(9), e1001105-e1001105 (2010-09-15)
The essential coenzyme nicotinamide adenine dinucleotide (NAD+) plays important roles in metabolic reactions and cell regulation in all organisms. Bacteria, fungi, plants, and animals use different pathways to synthesize NAD+. Our molecular and genetic data demonstrate that in the unicellular
Iran Goudarzi et al.
European journal of pharmacology, 642(1-3), 56-65 (2010-07-20)
Electrophysiological dysfunction of Purkinje cells causes cerebellar ataxia. Recent studies indicated that 4-aminopyridine (4-AP) can prevent the attacks in patients with episodic ataxia type 2. However, the cellular mechanism(s) by which 4-AP might be beneficial for the improvement of motor
Jun Ho Song et al.
Cell reports, 31(8), 107682-107682 (2020-05-28)
Recent breakthroughs in neuroanatomical tracing methods have helped unravel complicated neural connectivity in whole-brain tissue at single-cell resolution. However, in most cases, analysis of brain images remains dependent on highly subjective and sample-specific manual processing, preventing precise comparison across sample
T M Kuchmerovskaia et al.
Voprosy meditsinskoi khimii, 48(3), 264-270 (2002-09-24)
The effect of administration of 3-acetylpyridine (antivitamin B3), on rat brain serotoninergic and NAD receptor systems was investigated. The injection of 3-acetylpyridine to rats caused a decrease of NAD and serotonin content in the brain and changes in [U-14C]NAD binding
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