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N7778

Sigma-Aldrich

7-Nitroindazol

≥98%

Synonym(e):

7-NI

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About This Item

Empirische Formel (Hill-System):
C7H5N3O2
CAS-Nummer:
2942-42-9
Molekulargewicht:
163.13
EG-Nummer:
220-934-4
MDL-Nummer:
MFCD00022789
UNSPSC-Code:
12352200
PubChem Substanz-ID:
24278047
NACRES:
NA.77

Qualitätsniveau

100

Assay

≥98%

Form

powder

Löslichkeit

DMF: 50 mg/mL, clear, yellow to orange

Lagertemp.

−20°C

SMILES String

[O-][N+](=O)c1cccc2cn[nH]c12

InChI

1S/C7H5N3O2/c11-10(12)6-3-1-2-5-4-8-9-7(5)6/h1-4H,(H,8,9)

InChIKey

PQCAUHUKTBHUSA-UHFFFAOYSA-N

Angaben zum Gen

human ... NOS1(4842) , NOS2(4843) , NOS2B(201288) , NOS2C(645740) , NOS3(4846)
rat ... Nos1(24598)

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Biochem./physiol. Wirkung

Selective inhibitor of nitric oxide synthase from mouse brain.

Leistungsmerkmale und Vorteile

This compound is featured on the Nitric Oxide Synthases page of the Handbook of Receptor Classification and Signal Transduction. To browse other handbook pages, click here.

Piktogramme

Skull and crossbonesHealth hazard
GHS06,GHS08

Signalwort

Danger

Gefahreneinstufungen

Acute Tox. 3 Oral - Carc. 2 - Eye Irrit. 2 - Repr. 1B

Lagerklassenschlüssel

6.1C - Combustible acute toxic Cat.3 / toxic compounds or compounds which causing chronic effects

WGK

WGK 3

Flammpunkt (°F)

Not applicable

Flammpunkt (°C)

Not applicable

Persönliche Schutzausrüstung

Eyeshields, Faceshields, Gloves, type P3 (EN 143) respirator cartridges

Analysenzertifikate (COA)

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1-Phenylimidazol 97%

Sigma-Aldrich

357774

1-Phenylimidazol

Diphenyleniodoniumchlorid A cell-permeable, irreversible inhibitor of endothelial nitric oxide synthase (eNOS).

Sigma-Aldrich

300260

Diphenyleniodoniumchlorid

Diphenyleniodoniumchlorid ≥98%

Sigma-Aldrich

D2926

Diphenyleniodoniumchlorid

Philippe Bonnin et al.
International journal of molecular sciences, 21(18) (2020-09-12)
The poly(ADP-ribose) polymerase (PARP) inhibitor PJ34 has been reported to improve endothelial dysfunction in the peripheral system. We addressed the role of PJ34 on the vascular tone and vasoreactivity during development in the mouse brain. Blood flows were measured in
J Garthwaite
Trends in neurosciences, 14(2), 60-67 (1991-02-01)
Nitric oxide (NO) is a recently discovered and highly unorthodox messenger molecule. Current evidence indicates that, in the CNS, NO is produced enzymatically in postsynaptic structures in response to activation of excitatory amino acid receptors. It then diffuses out to
Dongmei Wang et al.
Experimental neurology, 239, 111-119 (2012-10-16)
We have previously shown that intrathecal administration of the adrenomedullin (AM) receptor antagonist AM(22-52) produces a long-lasting anti-hyperalgesia effect. This study examined the hypothesis that AM recruits other pronociceptive mediators in complete Freund's adjuvant (CFA)-induced inflammation. Injection of CFA in
Morten Hedetoft et al.
Undersea & hyperbaric medicine : journal of the Undersea and Hyperbaric Medical Society, Inc, 45(3), 335-350 (2018-07-22)
Experiments have shown that hyperbaric oxygen (HBO2) therapy reduces cyanide-induced cerebral distress. The exact mechanism behind HBO2's neuroprotective effect is unknown, but has been proposed to be mediated by an increased neuronal nitric oxide (NO) bioavailability, which may compete with
Anita J Turner et al.
American journal of physiology. Renal physiology, 313(4), F864-F873 (2017-07-07)
Via developmental programming, prenatal perturbations, such as exposure to glucocorticoids and maternal malnutrition alter kidney development and contribute to the development of hypertension. To examine the possibility that alterations in tubuloglomerular feedback (TGF) contribute to the development of hypertension in
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