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61251

Sigma-Aldrich

Palmitoyl-L-carnitin

≥97.0% (HPLC)

Synonym(e):

(2R)-3-Carboxy-N,N,N-trimethyl-2-[(1-oxohexadecyl)-oxy]-1-propanaminium Inneres Salz, L-Carnitin-hexadecanoylester, C16-Carnitin, Hexadecanoyl-L-carnitin

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About This Item

Empirische Formel (Hill-System):
C23H45NO4
CAS-Nummer:
2364-67-2
Molekulargewicht:
399.61
Beilstein:
4152034
UNSPSC-Code:
12352211
PubChem Substanz-ID:
329759410
NACRES:
NA.26

Qualitätsniveau

100

Assay

≥97.0% (HPLC)

Optische Aktivität

[α]/D -17±2°, c = 1 in methanol

Verunreinigungen

≤10% water

Lagertemp.

2-8°C

SMILES String

C[N+](C)(C)C[C@H](OC(CCCCCCCCCCCCCCC)=O)CC([O-])=O

InChI

1S/C23H45NO4/c1-5-6-7-8-9-10-11-12-13-14-15-16-17-18-23(27)28-21(19-22(25)26)20-24(2,3)4/h21H,5-20H2,1-4H3/t21-/m1/s1

InChIKey

XOMRRQXKHMYMOC-OAQYLSRUSA-N

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Biochem./physiol. Wirkung

L-Palmitoylcarnitine can change the activity of several enzymes and transporters, localized in the membrane and facilitates the transfer of long-chain fatty acids from cytoplasm into mitochondria during the oxidation of fatty acids. L-Palmitoylcarnitine accumulates in ischemic myocardium and potentially contribute to myocardial damage through alterations in membrane molecular dynamics.

Lagerklassenschlüssel

11 - Combustible Solids

WGK

WGK 3

Flammpunkt (°F)

Not applicable

Flammpunkt (°C)

Not applicable

Analysenzertifikate (COA)

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Sanne J C M Frambach et al.
Biochimica et biophysica acta. Molecular basis of disease, 1866(6), 165727-165727 (2020-02-20)
Mitochondrial complex I (CI), the first multiprotein enzyme complex of the OXPHOS system, executes a major role in cellular ATP generation. Consequently, dysfunction of this complex has been linked to inherited metabolic disorders, including Leigh disease (LD), an often fatal
Katherine A Overmyer et al.
Cell metabolism, 21(3), 468-478 (2015-03-05)
Maximal exercise-associated oxidative capacity is strongly correlated with health and longevity in humans. Rats selectively bred for high running capacity (HCR) have improved metabolic health and are longer-lived than their low-capacity counterparts (LCR). Using metabolomic and proteomic profiling, we show
Stephanie J Mihalik et al.
Obesity (Silver Spring, Md.), 18(9), 1695-1700 (2010-01-30)
Dysregulation of fatty acid oxidation (FAO) is recognized as important in the pathophysiology of obesity and insulin resistance (IR). However, demonstrating FAO defects in vivo in humans has entailed complex and invasive methodologies. Recently, the identification of genetic blocks in
Margaret-Ann M Nelson et al.
Amino acids, 51(1), 97-102 (2018-09-08)
Oxidative deamination of norepinephrine (NE) and dopamine (DA) by monoamine oxidase (MAO) generates the catecholaldehydes 3,4-dihydroxyphenylglycolaldehyde (DOPEGAL) and 3,4-dihydroxyphenylacetaldehyde (DOPAL), respectively, and H2O2. Catecholaldehydes are highly reactive electrophiles that have been implicated as causal factors in the etiology of neurodegenerative
Ji Young Kim et al.
Journal of proteome research, 9(9), 4368-4375 (2010-06-22)
Obesity is currently epidemic in many countries worldwide and is strongly related to diabetes and cardiovascular disease. This study investigated the differences in metabolomic profiling between overweight/obese and normal-weight men. Overweight/obese (n=30) and age-matched, normal-weight men (n=30) were included. Anthropometric
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