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S0882

Sigma-Aldrich

Natriumselenat

≥95% (elemental analysis)

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About This Item

Lineare Formel:
Na2SeO4
CAS-Nummer:
13410-01-0
Molekulargewicht:
188.94
EG-Nummer:
236-501-8
MDL-Nummer:
MFCD00003490
UNSPSC-Code:
12352302
PubChem Substanz-ID:
329824181
NACRES:
NA.55

Qualitätsniveau

200

Assay

≥95% (elemental analysis)

Form

powder or crystals

pH-Wert

5.5-7.5 (25 °C, 18.9 g/L)

SMILES String

[Na+].[Na+].[O-][Se]([O-])(=O)=O

InChI

1S/2Na.H2O4Se/c;;1-5(2,3)4/h;;(H2,1,2,3,4)/q2*+1;/p-2

InChIKey

MHQOTKLEMKRJIR-UHFFFAOYSA-L

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Anwendung


  • Selenium nanoparticles alleviate ischemia reperfusion injury-induced acute kidney injury by modulating GPx-1/NLRP3/Caspase-1 pathway.: This study demonstrates that selenium nanoparticles, including sodium selenate, mitigate acute kidney injury caused by ischemia reperfusion. The nanoparticles modulate key pathways, offering potential therapeutic applications in renal injury treatment (Wang et al., 2022).

  • Phosphoproteomic profiling of selenate-treated Alzheimer′s disease model cells.: This research provides a detailed phosphoproteomic profile of Alzheimer′s disease model cells treated with sodium selenate, uncovering its effects on protein phosphorylation and implications for neuroprotection (Chen et al., 2014).

Signalwort

Danger

Gefahreneinstufungen

Acute Tox. 2 Inhalation - Acute Tox. 2 Oral - Aquatic Acute 1 - Aquatic Chronic 1 - Skin Irrit. 2 - STOT RE 2

Lagerklassenschlüssel

6.1B - Non-combustible acute toxic Cat. 1 and 2 / very toxic hazardous materials

WGK

WGK 2

Flammpunkt (°F)

Not applicable

Flammpunkt (°C)

Not applicable

Analysenzertifikate (COA)

Suchen Sie nach Analysenzertifikate (COA), indem Sie die Lot-/Chargennummer des Produkts eingeben. Lot- und Chargennummern sind auf dem Produktetikett hinter den Wörtern ‘Lot’ oder ‘Batch’ (Lot oder Charge) zu finden.

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Selen-L-methionin ≥98% (TLC), powder

Sigma-Aldrich

S3132

Selen-L-methionin

Selendioxid ≥99.9% trace metals basis

Sigma-Aldrich

200107

Selendioxid

Putrescin analytical standard

Supelco

51799

Putrescin

Natriumarsenat, zweibasisch Heptahydrat ≥98.0%

Sigma-Aldrich

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Natriumarsenat, zweibasisch Heptahydrat

Transferrin human powder, BioReagent, suitable for cell culture

Sigma-Aldrich

T8158

Transferrin human

W C Stewart et al.
Journal of animal science, 90(2), 577-584 (2011-10-04)
Adequate Se transfer from ewes to lambs is important to prevent Se-deficiency diseases. To evaluate how different chemical forms of Se administered at comparative dosages to mature ewes affect Se status of their lambs, 240 ewes were divided into 8
Beatrice Pezzarossa et al.
Journal of the science of food and agriculture, 92(4), 781-786 (2011-09-29)
We investigated the effects of the foliar and fruit application of sodium selenate on selenium (Se) accumulation, fruit growth and ripening in peach and pear. Trials were conducted in two growing seasons. In 2008 selenate was applied at a rate
Nigel C Jones et al.
Neurobiology of disease, 45(3), 897-901 (2011-12-21)
Tau hyperphosphorylation has been implicated in the pathogenesis of a variety of forms of human epilepsy. Here we investigated whether treatment with sodium selenate, a drug which reduces pathological hyperphosphorylated tau by enhancement of PP2A activity, would inhibit seizures in
N M Corcoran et al.
British journal of cancer, 103(4), 462-468 (2010-07-22)
Angiogenesis is fundamental to the progression of many solid tumours including prostate cancer. Sodium selenate is a small, water-soluble, orally bioavailable activator of PP2A phosphatase with anti-angiogenic properties. This was a dose-escalation phase I study in men with asymptomatic, chemotherapy-naïve
Janet van Eersel et al.
Proceedings of the National Academy of Sciences of the United States of America, 107(31), 13888-13893 (2010-07-21)
Alzheimer's disease (AD) brains are characterized by amyloid-beta-containing plaques and hyperphosphorylated tau-containing neurofibrillary tangles (NFTs); however, in frontotemporal dementia, the tau pathology manifests in the absence of overt amyloid-beta plaques. Therapeutic strategies so far have primarily been targeting amyloid-beta, although
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