Journal of environmental science and health. Part. B, Pesticides, food contaminants, and agricultural wastes, 20(5), 559-575 (1985-10-01)
In-vitro inhibitions of brain AChE in brook trout, Salvelinus fontinalis (Mitchill), by aminocarb (4-dimethylamino-m-tolyl N-methylcarbamate) and its toxic metabolites, MAA (4-methylamino-m-tolyl N-methylcarbamate), AA(4-amino-m-tolyl N-methylcarbamate), MFA (4-methylformamido-m-tolyl N-methylcarbamate) and FA (4-formamido-m-tolyl N-methylcarbamate) were investigated. The molar concentrations of inhibitors causing 50%
Journal of toxicology and environmental health, 20(1-2), 209-218 (1987-01-01)
The dermal penetration of 14C-ring-labeled fenitrothion and aminocarb was determined in rats and rhesus monkeys. In monkeys, 49 +/- 4% (t1/2 = 14 h) of the fenitrothion and 74 +/- 4% (t1/2 = 25 h) of aminocarb were absorbed from
Brain ChE response in forest songbirds exposed to aerial spraying of aminocarb and possible influence of application methodology and insecticide formulation.
D G Busby et al.
Bulletin of environmental contamination and toxicology, 31(2), 125-131 (1983-08-01)
Measurement of aminocarb in long-distance drift following aerial application to forests.
C M Riley et al.
Bulletin of environmental contamination and toxicology, 42(1), 37-44 (1989-01-01)
Ecotoxicology and environmental safety, 12(2), 180-186 (1986-10-01)
Young brown bullhead (Ictalurus nebulosus) were exposed to aminocarb (4-dimethylamino-3-methylphenyl N-methylcarbamate) at lethal and sublethal concentrations and the tissue distribution of total unspecified residues was examined. The concentration of residues in each tissue increased with the concentration of exposure. The
