추천 제품
생물학적 소스
goat
Quality Level
항체 형태
serum
항체 생산 유형
primary antibodies
클론
polyclonal
양식
liquid
미포함
preservative
종 반응성(상동성에 의해 예측)
all
제조업체/상표
Calbiochem®
저장 조건
OK to freeze
avoid repeated freeze/thaw cycles
동형
IgG
배송 상태
wet ice
저장 온도
−20°C
타겟 번역 후 변형
unmodified
일반 설명
Goat polyclonal antibody supplied as undiluted serum. Recognizes 8-hydroxydeoxy-guanosine.
Recognizes 8-Hydroxydeoxyguanosine in Alzheimer′s disease brain tissue.
This Anti-8-Hydroxyguanosine Goat polyclonal antibody detects 8-Hydroxydeoxyguanosine in Alzheimer′s disease brain tissue. It is validated for use in ELISA and with paraffin sections.
면역원
8-Hydroxyguanosine
애플리케이션
ELISA (≥1:250,000)
Paraffin Sections (≥1:200, proteinase K pre-treatment recommended; see figure legend, see application references)
경고
Toxicity: Standard Handling (A)
물리적 형태
Undiluted serum.
재구성
Following initial thaw, aliquot and freeze (-20°C).
분석 메모
Positive Control
Alzheimer’s disease brain tissue
Alzheimer’s disease brain tissue
기타 정보
Abe, T., et al. 2003. Neurosci. Lett.336, 105.
Parker, A.R., et al. 2002. Cancer Res.62, 7230.
Parker, A.R., et al. 2002. Cancer Res.62, 7230.
Does not cross-react with other naturally occurring nucleotides. Antibody should be titrated for optimal results in individual systems.
법적 정보
CALBIOCHEM is a registered trademark of Merck KGaA, Darmstadt, Germany
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Storage Class Code
10 - Combustible liquids
WGK
WGK 1
Flash Point (°F)
Not applicable
Flash Point (°C)
Not applicable
시험 성적서(COA)
제품의 로트/배치 번호를 입력하여 시험 성적서(COA)을 검색하십시오. 로트 및 배치 번호는 제품 라벨에 있는 ‘로트’ 또는 ‘배치’라는 용어 뒤에서 찾을 수 있습니다.
N Gueven et al.
Cell death and differentiation, 14(6), 1149-1161 (2007-03-10)
Several different autosomal recessive genetic disorders characterized by ataxia with oculomotor apraxia (AOA) have been identified with the unifying feature of defective DNA damage recognition and/or repair. We describe here the characterization of a novel form of AOA showing increased
Joseph J Boyle et al.
The American journal of pathology, 174(3), 1097-1108 (2009-02-24)
Intraplaque hemorrhage accelerates atherosclerosis via oxidant stress and contributes to lesion development and destabilization. Normally, macrophages scavenge hemoglobin-haptoglobin (HbHp) complexes via CD163, and this process provokes the secretion of the anti-inflammatory atheroprotective cytokine interleukin (IL)-10. We therefore tested the hypothesis
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