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isotopic purity
95 atom % 11B
form
solid
mass shift
depleted
SMILES string
[11B]
InChI
1S/B/i1+0
InChI key
ZOXJGFHDIHLPTG-IGMARMGPSA-N
Packaging
This product may be available from bulk stock and can be packaged on demand. For information on pricing, availability and packaging, please contact Stable Isotopes Customer Service.
Signal Word
Warning
Hazard Statements
Precautionary Statements
Hazard Classifications
Acute Tox. 4 Oral - Aquatic Chronic 3
Storage Class Code
11 - Combustible Solids
WGK
WGK 3
Flash Point(F)
Not applicable
Flash Point(C)
Not applicable
Certificates of Analysis (COA)
Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.
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Cell death & disease, 9(10), 967-967 (2018-09-22)
Bcl-2 family proteins play a crucial role in regulating apoptosis, a process critical for development, eliminating damaged or infected cells, host-pathogen interactions and in disease. Dysregulation of Bcl-2 proteins elicits an expansive cell survival mechanism promoting cell migration, invasion and
Nature methods, 14(9), 891-896 (2017-07-25)
We developed a chemically inducible Cas9 (ciCas9) and a droplet digital PCR assay for double-strand breaks (DSB-ddPCR) to investigate the kinetics of Cas9-mediated generation and repair of DSBs in cells. ciCas9 is a rapidly activated, single-component Cas9 variant engineered by
Physiological reviews, 98(2), 813-880 (2018-03-01)
Neuronal cell death occurs extensively during development and pathology, where it is especially important because of the limited capacity of adult neurons to proliferate or be replaced. The concept of cell death used to be simple as there were just
Polymers, 11(5) (2019-05-11)
A series of fluorenyl-based constrained-geometry-configuration (CGC) allyl-type rare earth metal monoalkyl complexes bearing the divalent anionic η3:η1-tert-butyl(dimethylfluorenylsilyl)amido (η3:η1-FluSiMe2N
PLoS biology, 10(9), e1001394-e1001394 (2012-10-11)
Bax/Bak-mediated mitochondrial outer membrane permeabilization (MOMP) is essential for "intrinsic" apoptotic cell death. Published studies used synthetic liposomes to reveal an intrinsic pore-forming activity of Bax, but it is unclear how other mitochondrial outer membrane (MOM) proteins might facilitate this
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