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About This Item
Linear Formula:
CH3(CH2)7NHC(=NH)NH2 · 1/2H2SO4
CAS Number:
Molecular Weight:
220.32
MDL number:
UNSPSC Code:
12352100
PubChem Substance ID:
NACRES:
NA.22
Recommended Products
Assay
97%
SMILES string
OS(O)(=O)=O.CCCCCCCCNC(N)=N.CCCCCCCCNC(N)=N
InChI
1S/2C9H21N3.H2O4S/c2*1-2-3-4-5-6-7-8-12-9(10)11;1-5(2,3)4/h2*2-8H2,1H3,(H4,10,11,12);(H2,1,2,3,4)
InChI key
DTTVLDRWEFQROK-UHFFFAOYSA-N
General description
1-Octylguanidine hemisulfate is a guanidine derivative. Octyl guanidine sulfate is prepared from S-methylisothiourea sulfate and octylamine.
Signal Word
Warning
Hazard Statements
Precautionary Statements
Hazard Classifications
Eye Irrit. 2 - Skin Irrit. 2 - STOT SE 3
Target Organs
Respiratory system
Storage Class Code
11 - Combustible Solids
WGK
WGK 3
Flash Point(F)
Not applicable
Flash Point(C)
Not applicable
Personal Protective Equipment
dust mask type N95 (US), Eyeshields, Gloves
Certificates of Analysis (COA)
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Natalia Pavón et al.
Journal of biochemistry, 149(2), 211-217 (2010-11-30)
Mercurials are known to induce morphological and functional modifications in kidney. The protective effect of octylguanidine on the injury induced by Hg(2+) on renal functions was studied. Octylguanidine administered at a dose of 10 mg/kg body weight prevented the damage
J R de Weille et al.
Brain research, 445(1), 1-11 (1988-03-29)
Kinetics of pyrethroid-modified sodium channels and the interaction of N-octylguanidine with the modified channels have been studied with internally perfused and voltage-clamped squid giant axons. The pyrethroids used were 1R-cis-phenothrin; 1R-cis-permethrin; 1R-cis-cyphenothrin; and 1R-cis-deltamethrin. Modification of sodium channels by pyrethroids
Studies on nitrogen metabolism in tobacco plants IX. Effect of various compounds on proline biosynthesis in the green leaves.
Noguchi M, et al.
Plant & Cell Physiology, 9(1), 35-47 (1968)
Victoriano Pérez-Vázquez et al.
Journal of bioenergetics and biomembranes, 35(3), 231-241 (2003-09-19)
The yeast mitochondrial unspecific channel (YMUC) sensitivity to inorganic (Ca2+ or Mg2+) or organic (hexyl or octyl-guanidine) cations was measured. The rate of oxygen consumption in State 3 and State 4, the transmembrane potential (deltapsi), mitochondrial swelling, and the polyethylene-glycol
Natalia Pavón et al.
Endocrine, 35(2), 158-165 (2009-01-27)
Hyperthyroidism sensitizes the heart for reperfusion injury. As known, mitochondrial permeability transition underlies reperfusion heart damage. This study was undertaken to explore the protective effect of octylguanidine (OG), an inhibitor of permeability transition, on hearts from hyperthyroid rats subjected to
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