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SAB3701153

Sigma-Aldrich

Anti-Mouse IgG (H+L), F(ab′)2 fragment, highly cross adsorbed-Biotin antibody produced in donkey

affinity isolated antibody, lyophilized powder

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About This Item

Codice UNSPSC:
12352203
NACRES:
NA.46

Origine biologica

donkey

Coniugato

biotin conjugate

Forma dell’anticorpo

affinity isolated antibody

Tipo di anticorpo

secondary antibodies

Clone

polyclonal

Forma fisica

lyophilized powder

Reattività contro le specie

mouse

tecniche

immunohistochemistry: suitable
indirect ELISA: suitable
western blot: suitable

Condizioni di spedizione

wet ice

Temperatura di conservazione

2-8°C

modifica post-traduzionali bersaglio

unmodified

Categorie correlate

Specificità

This product was prepared from monospecific antiserum by immunoaffinity chromatography using Mouse IgG coupled to agarose beads followed by solid phase adsorption(s) to remove any unwanted reactivities, pepsin digestion and chromatographic separation. Assay by immunoelectrophoresis resulted in a single precipitin arc against Anti-Biotin, Anti-Donkey Serum, Mouse IgG and Mouse Serum. No reaction was observed against Anti-Pepsin, Anti-Donkey IgG F(c) or Bovine, Chicken, Goat, Guinea Pig, Hamster, Horse, Human, Rabbit, Rat and Sheep Serum Proteins.

Immunogeno

Mouse IgG whole molecule

Proprietà fisiche

Antibody format: IgG F(ab′)2

Stato fisico

Supplied in 0.02 M Potassium Phosphate, 0.15 M Sodium Chloride, pH 7.2 with 10 mg/mL Bovine Serum Albumin (BSA) - Immunoglobulin and Protease free

Ricostituzione

Reconstitute with 500 μ;L deionized water (or equivalent).

Esclusione di responsabilità

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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Pittogrammi

Skull and crossbonesEnvironment
GHS06,GHS09

Avvertenze

Danger

Indicazioni di pericolo

H302,H311,H411

Classi di pericolo

Acute Tox. 3 Dermal - Acute Tox. 4 Oral - Aquatic Chronic 2

Rischi supp

EUH032

Codice della classe di stoccaggio

6.1C - Combustible acute toxic Cat.3 / toxic compounds or compounds which causing chronic effects

Classe di pericolosità dell'acqua (WGK)

WGK 3

Punto d’infiammabilità (°F)

Not applicable

Punto d’infiammabilità (°C)

Not applicable

Certificati d'analisi (COA)

Cerca il Certificati d'analisi (COA) digitando il numero di lotto/batch corrispondente. I numeri di lotto o di batch sono stampati sull'etichetta dei prodotti dopo la parola ‘Lotto’ o ‘Batch’.

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Experimental eye research, 165, 78-89 (2017-09-11)
Müller cells, the supporting cells of the retina, play a key role in responding to retinal stress by releasing chemokines, including CCL2, to recruit microglia and macrophages (MG/MΦ) into the damaged retina. Photobiomodulation (PBM) with 670 nm light has been shown
Gu-Choul Shin et al.
Experimental & molecular medicine, 56(1), 192-209 (2024-01-11)
Tumor necrosis factor superfamily (TNFSF) resistance contributes to the development and progression of tumors and resistance to various cancer therapies. Tumor-intrinsic alterations involved in the adaptation to the TNFSF response remain largely unknown. Here, we demonstrate that protein kinase C
Gu-Choul Shin et al.
Nature communications, 10(1), 3185-3185 (2019-07-20)
Unfolded protein response (UPR) is an adaptive mechanism that aims at restoring ER homeostasis under severe environmental stress. Malignant cells are resistant to environmental stress, which is largely due to an activated UPR. However, the molecular mechanisms by which different
Gu-Choul Shin et al.
Autophagy, 12(12), 2451-2466 (2016-10-30)
Death receptors of TNFSF10/TRAIL (tumor necrosis factor superfamily member 10) contribute to immune surveillance against virus-infected or transformed cells by promoting apoptosis. Many viruses evade antiviral immunity by modulating TNFSF10 receptor signaling, leading to persistent infection. Here, we report that

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