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Documenti fondamentali

AB907

Sigma-Aldrich

Anti-Desmin Antibody

serum, Chemicon®

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About This Item

Codice UNSPSC:
12352203
eCl@ss:
32160702
NACRES:
NA.41
Clone:
polyclonal
application:
IHC
WB
Reattività contro le specie:
mouse, bovine, chicken, human
tecniche:
immunohistochemistry: suitable (paraffin)
western blot: suitable
citations:
10

Origine biologica

rabbit

Livello qualitativo

Forma dell’anticorpo

serum

Tipo di anticorpo

primary antibodies

Clone

polyclonal

Reattività contro le specie

mouse, bovine, chicken, human

Produttore/marchio commerciale

Chemicon®

tecniche

immunohistochemistry: suitable (paraffin)
western blot: suitable

N° accesso NCBI

N° accesso UniProt

Condizioni di spedizione

dry ice

modifica post-traduzionali bersaglio

unmodified

Informazioni sul gene

human ... DES(1674)

Specificità

Desmin. Reacts specifically with desmin in cultured cells or tissue preparations originating from chicken, human, bovine and mouse tissue. AB907 specifically stains the wide desmin band in immunoblot at a molecular weight of 50-55 kD.

Immunogeno

Purified desmin from chicken gizzard, isolated by modified procedure of Geisler, et al [J. Biol Chem. (1980). 111:425-433].

Applicazioni

Anti-Desmin Antibody is an antibody against Desmin for use in WB, IH(P).
Indirect immunofluorescence of formalin fixed paraffin-embedded sections of human intestine sections: 1:10-1:20.

Immunoblotting

Optimal working dilutions must be determined by the end user.
Research Category
Cell Structure
Research Sub Category
Cytoskeleton

Linkage

Replaces: 04-585

Stato fisico

Rabbit antiserum. Liquid containing 0.1% sodium azide as a preservative.

Stoccaggio e stabilità

Maintain at -20°C in undiluted aliquots for up to 12 months. Avoid repeated freeze/thaw cycles.

Note legali

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

Esclusione di responsabilità

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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Raccomandato

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Descrizione
Determinazione del prezzo

Codice della classe di stoccaggio

10 - Combustible liquids

Classe di pericolosità dell'acqua (WGK)

WGK 1


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Yee Kit Tai et al.
Cells, 13(5) (2024-03-13)
Briefly (10 min) exposing C2C12 myotubes to low amplitude (1.5 mT) pulsed electromagnetic fields (PEMFs) generated a conditioned media (pCM) that was capable of mitigating breast cancer cell growth, migration, and invasiveness in vitro, whereas the conditioned media harvested from
Federica Maione et al.
Methods in molecular biology (Clifton, N.J.), 1267, 349-365 (2015-02-01)
Angiogenesis, the formation of neo-vessels, is one of the most important hallmarks of cancer. Tumor vasculature presents structural abnormalities such as dilatation of vessel diameter and hyper-branched and twisted pattern. A promising strategy in anticancer therapy to overcome the resistance
Witold W Kilarski et al.
Laboratory investigation; a journal of technical methods and pathology, 85(5), 643-654 (2005-02-22)
Migration, proliferation and invasive growth of myofibroblasts are key cellular events during formation of granulation tissue in situations of wound healing, arteriosclerosis and tumor growth. To study the invasive phenotype of myofibroblasts, we established an assay where arterial tissue from
G P Sui et al.
BJU international, 90(1), 118-129 (2002-06-26)
To determine whether suburothelial interstitial cells of the human bladder express gap junctions, and if so, to establish their extent and composition, using immunocytochemistry, confocal microscopy and electron microscopy. Bladder tissue was obtained at cystectomy; the tissue was: (i) frozen
Richard L Benton et al.
The Journal of comparative neurology, 507(1), 1031-1052 (2007-12-20)
After traumatic spinal cord injury (SCI), disruption and plasticity of the microvasculature within injured spinal tissue contribute to the pathological cascades associated with the evolution of both primary and secondary injury. Conversely, preserved vascular function most likely results in tissue

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