GF53990253
Iron
foil, 8mm disks, thickness 0.5mm, hard, 99.5%
Sinonimo/i:
Iron, FE000405
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About This Item
Formula condensata:
Fe
Numero CAS:
Peso molecolare:
55.85
Numero MDL:
Codice UNSPSC:
12141721
ID PubChem:
NACRES:
NA.23
Saggio
99.5%
Forma fisica
foil
Produttore/marchio commerciale
Goodfellow 539-902-53
Resistività
9.71 μΩ-cm
diam. × spessore
8 mm × 0.5 mm
P. eboll.
2750 °C (lit.)
Punto di fusione
1535 °C (lit.)
Densità
7.86 g/mL at 25 °C (lit.)
Stringa SMILE
[Fe]
InChI
1S/Fe
XEEYBQQBJWHFJM-UHFFFAOYSA-N
Categorie correlate
Descrizione generale
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Jun-Won Jang et al.
Water science and technology : a journal of the International Association on Water Pollution Research, 59(12), 2503-2507 (2009-06-23)
Zero valent iron has been successfully used for the degradation of a wide range of contaminants. However, this reaction of using ZVI particle produces a large quantity of iron sludge. To solve the problem, we report the synthesis of self-organized
Arnold L Demain et al.
Applied microbiology and biotechnology, 73(1), 55-59 (2006-04-20)
When tetanus toxin is made by fermentation with Clostridium tetani, the traditional source of iron is an insoluble preparation called reduced iron powder. This material removes oxygen from the system by forming FeO(2) (rust). When inoculated in a newly developed
50 years ago in the Journal of Pediatrics: Iron metabolism in premature infants: II. Prevention of iron deficiency.
Pamela J Kling
The Journal of pediatrics, 164(4), 768-768 (2014-03-22)
Thomas A Russo et al.
Infection and immunity, 82(6), 2356-2367 (2014-03-26)
Hypervirulent (hypermucoviscous) Klebsiella pneumoniae (hvKP) strains are an emerging variant of "classical" K. pneumoniae (cKP) that cause organ and life-threatening infection in healthy individuals. An understanding of hvKP-specific virulence mechanisms that enabled evolution from cKP is limited. Observations by our
Eunna Choi et al.
Infection and immunity, 82(6), 2203-2210 (2014-03-20)
In order to survive inside macrophages, Salmonella produces a series of proteins encoded by genes within Salmonella pathogenicity island 2 (SPI-2). In the present study, we report that Fur, a central regulator of iron utilization, negatively controls the expression of
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