GF17563293
Copper - O.F.H.C.
foil, 300x1000mm, thickness 2.0mm, half hard, 99.95+%
Sinonimo/i:
Copper, CV000765
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About This Item
Formula empirica (notazione di Hill):
Cu
Numero CAS:
Peso molecolare:
63.55
Numero MDL:
Codice UNSPSC:
12141711
ID PubChem:
NACRES:
NA.23
Saggio
99.95%
Forma fisica
foil
Produttore/marchio commerciale
Goodfellow 175-632-93
Resistività
1.673 μΩ-cm, 20°C
Misura × spessore
300 x 1000 mm × 2.0 mm
P. eboll.
2567 °C (lit.)
Punto di fusione
1083.4 °C (lit.)
Densità
8.94 g/mL at 25 °C (lit.)
Stringa SMILE
[Cu]
InChI
1S/Cu
RYGMFSIKBFXOCR-UHFFFAOYSA-N
Categorie correlate
Descrizione generale
For updated SDS information please visit www.goodfellow.com.
Note legali
Product of Goodfellow
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I documenti relativi ai prodotti acquistati recentemente sono disponibili nell’Archivio dei documenti.
Donita C Brady et al.
Nature, 509(7501), 492-496 (2014-04-11)
The BRAF kinase is mutated, typically Val 600→Glu (V600E), to induce an active oncogenic state in a large fraction of melanomas, thyroid cancers, hairy cell leukaemias and, to a smaller extent, a wide spectrum of other cancers. BRAF(V600E) phosphorylates and
Jonas Duus Stevens Lekfeldt et al.
Environmental pollution (Barking, Essex : 1987), 194, 78-85 (2014-08-06)
Copper (Cu) is known to accumulate in agricultural soils receiving urban waste products as fertilizers. We here report the use of the leucine incorporation technique to determine pollution-induced community tolerance (Leu-PICT) to Cu in a long-term agricultural field trial. A
Julie E Gleason et al.
Proceedings of the National Academy of Sciences of the United States of America, 111(16), 5866-5871 (2014-04-09)
The human fungal pathogens Candida albicans and Histoplasma capsulatum have been reported to protect against the oxidative burst of host innate immune cells using a family of extracellular proteins with similarity to Cu/Zn superoxide dismutase 1 (SOD1). We report here
Lelita T Braiterman et al.
Proceedings of the National Academy of Sciences of the United States of America, 111(14), E1364-E1373 (2014-04-08)
Wilson disease (WD) is a monogenic autosomal-recessive disorder of copper accumulation that leads to liver failure and/or neurological deficits. WD is caused by mutations in ATP7B, a transporter that loads Cu(I) onto newly synthesized cupro-enzymes in the trans-Golgi network (TGN)
Alina Fedoseienko et al.
Annals of the New York Academy of Sciences, 1314, 6-14 (2014-04-05)
Copper is an important cofactor in numerous biological processes in all living organisms. However, excessive copper can be extremely toxic, so it is vital that the copper level within a cell is tightly regulated. The damaging effect of copper is
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