推荐产品
等級
ACS reagent
化驗
98.0-101.0%
雜質
≤0.005% Insoluble matter
pH值
3.5-6.0 (25 °C, 5% in solution)
mp
58 °C (lit.)
負離子痕跡
sulfate (SO42-): ≤0.005%
正離子痕跡
Ca: ≤0.005%
Fe: ≤5 ppm
K: ≤0.01%
Mg: ≤0.005%
Na: ≤0.05%
Zn: ≤0.005%
heavy metals (as Pb): ≤5 ppm
儲存溫度
2-8°C
SMILES 字串
Cl[Mn]Cl.[H]O[H].[H]O[H].[H]O[H].[H]O[H]
InChI
1S/2ClH.Mn.4H2O/h2*1H;;4*1H2/q;;+2;;;;/p-2
InChI 密鑰
CNFDGXZLMLFIJV-UHFFFAOYSA-L
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訊號詞
Danger
危險聲明
危險分類
Acute Tox. 3 Oral - Eye Dam. 1 - STOT RE 2
標靶器官
Brain
儲存類別代碼
6.1D - Non-combustible acute toxic Cat.3 / toxic hazardous materials or hazardous materials causing chronic effects
水污染物質分類（WGK）
WGK 2
閃點（°F）
does not flash
閃點（°C）
does not flash
個人防護裝備
dust mask type N95 (US), Eyeshields, Gloves
Marta Sidoryk-Wegrzynowicz et al.
Journal of neurochemistry, 122(4), 856-867 (2012-06-20)
Manganese (Mn) has been implicated in the impairment of the glutamate-glutamine cycling (GGC) by deregulation of Glu and glutamine (Gln) turnover in astrocytes. Here, we have examined possible mechanisms involved in the Mn(II)-mediated disruption of Glu turnover, including those related
Katharina A Sterenczak et al.
BMC cancer, 12, 284-284 (2012-07-13)
Cell lines represent a key tool in cancer research allowing the generation of neoplasias which resemble initial tumours in in-vivo animal models. The characterisation of early tumour development is of major interest in order to evaluate the efficacy of therapeutic
Barry J Bowman et al.
Eukaryotic cell, 11(11), 1362-1370 (2012-09-18)
The pmr gene is predicted to encode a Ca(2+)-ATPase in the secretory pathway. We examined two strains of Neurospora crassa that lacked PMR: the Δpmr strain, in which pmr was completely deleted, and pmr(RIP), in which the gene was extensively
Jerome A Roth et al.
Toxicology in vitro : an international journal published in association with BIBRA, 26(7), 1143-1149 (2012-07-31)
Mutations in the parkin gene are linked to development of juvenile onset of Parkinson's disease and recent studies have reported that parkin can protect against increased oxidative stress and mitochondrial dysfunction caused by a variety of oxidative and toxic insults.
A G Kanthasamy et al.
Toxicology letters, 214(3), 288-295 (2012-09-22)
The role of normal cellular prion protein (PrP) remains to be fully elucidated; however, the protein is crucial for the infection and progression of prion diseases. Recent evidence indicates that PrP is a metalloprotein since the octapeptide repeat sequences in
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