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Merck

EMU146171

Sigma-Aldrich

MISSION® esiRNA

targeting mouse Ccr5

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About This Item

分類程式碼代碼:
41105324
NACRES:
NA.51

描述

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產品線

MISSION®

形狀

lyophilized powder

esiRNA cDNA 標靶序列

GAACCCCTAGGCTTAGTTAGGTTGAAATACCCATTGAGGAAACAGCAAATACAAAGGAAGAATAAAGAGTTTAGCCGGGAAGGTAGTCTCATTTTACAGCCGGAATATAATGTTATCTCAGGCTAGCATTTTGTTCCTGCCTTCAGACCTAAATCCTACCACACCGGGACTGTGAAACACCTGGATTATGAATCATGAGCCTGAGGTCTAGGAATAATAATAACGTTTGTGATTTTAGATGAGGGCTGTTTCCATAGTTTGAAGCCAGAACTTTATCATCTTGAGCAGAAGCTCCAAGAGATGAGGAAAGAGCACCAATTTTTCTCTAATTTACTTAGCAGTCATCATCTCTGGAAGATTCATTTTAGAAACAAGTTGTTGTGCCCCTCAGAAGCCATGAGAGTATAACGACTGCTCTCTGTGTTCCAGGCTGAGTATGAGGACTTCAGTCACACTTTCCAGATGGCTTCTCCACACAAACAATGCTAAGTTTGGCCATTTCAGAGGTTT

Ensembl | 小鼠類登錄號

NCBI登錄號

運輸包裝

ambient

儲存溫度

−20°C

基因資訊

相关类别

一般說明

MISSION® esiRNA are endoribonuclease prepared siRNA. They are a heterogeneous mixture of siRNA that all target the same mRNA sequence. These multiple silencing triggers lead to highly-specific and effective gene silencing.

For additional details as well as to view all available esiRNA options, please visit SigmaAldrich.com/esiRNA.

法律資訊

MISSION is a registered trademark of Merck KGaA, Darmstadt, Germany

儲存類別代碼

10 - Combustible liquids

閃點(°F)

Not applicable

閃點(°C)

Not applicable


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Shen Pang et al.
PloS one, 9(5), e96445-e96445 (2014-05-17)
The use of siRNAs to knock down gene expression can potentially be an approach to treat various diseases. To avoid siRNA toxicity the less transcriptionally active H1 pol III promoter, rather than the U6 promoter, was proposed for siRNA expression.
S Y Lee et al.
Journal of dental research, 94(12), 1715-1723 (2015-09-12)
Tooth movement by application of orthodontic biophysical force primarily reflects the role of soluble molecules released from the periodontal ligament (PDL). Thus far, many factors have been reported to be involved in orthodontic tooth movement (OTM), but key molecules that
Lanfu Zhao et al.
Acta biochimica et biophysica Sinica, 47(11), 890-898 (2015-09-24)
Glioblastoma (GBM) is the most prevalent malignant primary brain tumor in adults and exhibits a spectrum of aberrantly aggressive phenotype. Tumor cell proliferation and invasion are critically regulated by chemokines and their receptors. Recent studies have shown that the chemokine

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