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Merck
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主要文件

EMU092761

Sigma-Aldrich

MISSION® esiRNA

targeting mouse Psmd2

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About This Item

UNSPSC代码:
41105324
NACRES:
NA.51

描述

Powered by Eupheria Biotech

产品线

MISSION®

表单

lyophilized powder

esiRNA cDNA靶序列

GGCTCTCAAGTGGATTCTGCACGAATGAACCTAGCCTCCTCTTTTGTAAATGGCTTTGTGAATGCAGCCTTTGGTCAAGATAAACTACTGACTGATGATGGCAACAAATGGCTTTACAAGAACAAGGACCATGGGATGCTAAGTGCTGCTGCGTCTCTTGGCATGATTCTGCTGTGGGATGTGGATGGTGGCCTCACCCAGATTGACAAGTATCTGTACTCCTCTGAGGACTACATCAAGTCAGGAGCTCTCCTTGCCTGTGGTATCGTGAATTCTGGAGTCCGGAATGAGTGTGATCCTGCCCTGGCACTGCTTTCAGACTATGTTCTCCATAACAGCAATACCATGAGACTTGGCTCCATCTTTGGGCTAGGTTTGGCCTATGCTGGCTCTAATCGGGAAGATGTTCTAACACTGCTGCTACCTGTAATGGGAGATTCCAAGTCCAGCATGGAGGTGGCAGGTGTGACGGCTCTAGCTTGTGGGATGATAGCAGTGGGGTCCTG

基因组数据库 |小鼠登记号

ENSMUSG00000006998

NCBI登记号

NM_134101

运输

ambient

储存温度

−20°C

基因信息

mouse ... PSMD2(21762) , Psmd2(21762)

一般描述

MISSION® esiRNA are endoribonuclease prepared siRNA. They are a heterogeneous mixture of siRNA that all target the same mRNA sequence. These multiple silencing triggers lead to highly-specific and effective gene silencing.

For additional details as well as to view all available esiRNA options, please visit SigmaAldrich.com/esiRNA.

法律信息

MISSION is a registered trademark of Merck KGaA, Darmstadt, Germany

储存分类代码

10 - Combustible liquids

闪点(°F)

Not applicable

闪点(°C)

Not applicable

历史批次信息供参考:

分析证书(COA)

Lot/Batch Number
QCS

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Christoph Gerhardt et al.
The Journal of cell biology, 210(1), 115-133 (2015-07-08)
Mutations in RPGRIP1L result in severe human diseases called ciliopathies. To unravel the molecular function of RPGRIP1L, we analyzed Rpgrip1l(-/-) mouse embryos, which display a ciliopathy phenotype and die, at the latest, around birth. In these embryos, cilia-mediated signaling was
Peter Tsvetkov et al.
eLife, 4 (2015-09-04)
Proteasomes are central regulators of protein homeostasis in eukaryotes. Proteasome function is vulnerable to environmental insults, cellular protein imbalance and targeted pharmaceuticals. Yet, mechanisms that cells deploy to counteract inhibition of this central regulator are little understood. To find such

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