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Merck

EMU039841

Sigma-Aldrich

MISSION® esiRNA

targeting mouse Runx1

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About This Item

分類程式碼代碼:
41105324
NACRES:
NA.51

描述

Powered by Eupheria Biotech

產品線

MISSION®

形狀

lyophilized powder

esiRNA cDNA 標靶序列

TGATCCTATCCAGCCTTCCAACTACTGAAGCTGATTTTCAAGGCTACTTAAAAAAAAAAAAACTGCAGCAAACATTAATGGATTTCTGTTGTGTTTAAATTCTCTACAGATTGTATTGTAAATATTTTATGAAGTAGATCATATGTATATATTTATATATACGAGCACATACATTAGTAGCACAATCTTTTAAAGTTACGGCTCTTGCTTTTGAGAAACGAAGTGAGTTTTTCATGGTAAGAGGGGCGCTCTGTATGGAAGACACTCCTAAGTTTTGTATTTTGTTGAGACTCTAAACAAAACTGACCCGCAAGAAAGAAAAACAAACTGACAGGCAACTAACTGGTGGAACTTCCAAATCTAGTTTTTGCTGGTCATTTTTTGGTTGTTGTTGTTGTTCTGCGCATAAATATTTTAGGACGCGTATGGGAATTTTGCTCCGGGACCGTTTGTAATAGCCAAAGACTGAACTTCAACTCTCAAAGCGAGGCTCTGTTGGGCATTTGACTTTGA

Ensembl | 小鼠類登錄號

NCBI登錄號

運輸包裝

ambient

儲存溫度

−20°C

基因資訊

相关类别

一般說明

MISSION® esiRNA are endoribonuclease prepared siRNA. They are a heterogeneous mixture of siRNA that all target the same mRNA sequence. These multiple silencing triggers lead to highly-specific and effective gene silencing.

For additional details as well as to view all available esiRNA options, please visit SigmaAldrich.com/esiRNA.

法律資訊

MISSION is a registered trademark of Merck KGaA, Darmstadt, Germany

儲存類別代碼

10 - Combustible liquids

閃點(°F)

Not applicable

閃點(°C)

Not applicable


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Ken-ichi Takayama et al.
Oncotarget, 6(4), 2263-2276 (2014-12-30)
Androgen receptor (AR) signaling is essential for the development of prostate cancer. Here, we report that runt-related transcription factor (RUNX1) could be a key molecule for the androgen-dependence of prostate cancer. We found RUNX1 is a target of AR and
Gillian Browne et al.
Journal of cellular physiology, 230(10), 2522-2532 (2015-03-25)
Runx1 is a transcription factor essential for definitive hematopoiesis, and genetic abnormalities in Runx1 cause leukemia. Runx1 is functionally promiscuous and acts as either an oncogene or tumor suppressor gene in certain epithelial cancers. Recent evidence suggests that Runx1 is
A de Laurentiis et al.
Oncogene, 34(49), 6018-6028 (2015-04-22)
The t(12;21) translocation is the most common genetic rearrangement in childhood acute lymphoblastic leukemia (ALL) and gives rise to the TEL-AML1 fusion gene. Many studies on TEL-AML1 describe specific properties of the fusion protein, but a thorough understanding of its

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