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Merck
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主要文件

EHU111901

Sigma-Aldrich

MISSION® esiRNA

targeting human NLRX1

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About This Item

UNSPSC代码:
41105324
NACRES:
NA.51

描述

Powered by Eupheria Biotech

质量水平

100

产品线

MISSION®

表单

lyophilized powder

esiRNA cDNA靶序列

GGCGGTGTCAGAATACTGGTCAGTGATCCTCAGTGAAGTCCAGCGGAACCTCAATAGCTGGGATCGGGCCCGGGTTCAGCGACACCTTGAGCTCCTACTGCGGGATCTGGAAGATAGCCGGGGTGCCACCCTTAATCCTTGGCGCAAGGCCCAGCTGCTGCGAGTGGAGGGCGAGGTCAGGGCCCTCCTGGAGCAGCTGGGAAGCTCTGGAAGCTGAGACACTGGCGGCAGGCACCTAGCTATGTGACCACTGGCCCTAAACCTTTTCCCTCTGTGGCCTCCTGGCTTGCACTGCTCCCTCTAGAAAGATTCCTTCAGGTCTGGAGGCAGAGGAATGGGCATAGCTGAGCCAGTTGCCCTCCTAGGGCATGTTTGACCAGGACTGAGTCTGGAATCTCCAAGTTAAAGATGGTGAATCAATGCTTCGGGCTTGGAGATGGAACATGCCT

基因组数据库 |人类登记号

ENSG00000160703

NCBI登记号

NM_024618

运输

ambient

储存温度

−20°C

基因信息

human ... NLRX1(79671) , NLRX1(79671)

相关类别

一般描述

MISSION esiRNA are endoribonuclease prepared siRNA. They are a heterogeneous mixture of siRNA that all target the same mRNA sequence. These multiple silencing triggers lead to highly-specific and effective gene silencing.

For additional details as well as to view all available esiRNA options, please visit SigmaAldrich.com/esiRNA.

法律信息

MISSION is a registered trademark of Merck KGaA, Darmstadt, Germany

储存分类代码

10 - Combustible liquids

闪点(°F)

Not applicable

闪点(°C)

Not applicable

历史批次信息供参考:

分析证书(COA)

Lot/Batch Number
DOR

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Ding Ma et al.
International immunopharmacology, 71, 7-13 (2019-03-13)
Osteoarthritis (OA) is a chronic debilitating disease characterized by joint degeneration. Excessive chondrocyte apoptosis and inflammation contributes to articular cartilage destruction in OA pathology. Nucleotide-binding oligomerization domain (NOD)-like receptor X1 (NLRX1) has emerged as a critical regulator of inflammation that
Haiyan Yin et al.
Scientific reports, 7, 44311-44311 (2017-03-14)
Nucleotide-binding domain and leucine-rich-repeat-containing family member X1 (NLRX1), located in mitochondria, can recognize cytoplasmic pattern recognition receptors and is tightly related to reactive oxygen species (ROS) production, mitochondrial function, apoptosis and inflammation. The present study was designed to explore whether

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