所有图片(1)
About This Item
经验公式（希尔记法）:
C14H28O6
CAS号:
分子量:
292.37
Beilstein:
4864266
EC 号:
MDL编号:
UNSPSC代码:
12352200
PubChem化学物质编号:
描述
non-ionic
方案
~80% (TLC)
表单
paste
分子量
292.37 g/mol
缺失
≤10% loss on drying
颜色
beige
SMILES字符串
CCCCCCCCOC1O[C@H](CO)[C@@H](O)[C@H](O)[C@H]1O
InChI
1S/C14H28O6/c1-2-3-4-5-6-7-8-19-14-13(18)12(17)11(16)10(9-15)20-14/h10-18H,2-9H2,1H3/t10-,11-,12+,13-,14?/m1/s1
InChI key
HEGSGKPQLMEBJL-RQICVUQASA-N
其他说明
For the crystallisation of membrane proteins
储存分类代码
13 - Non Combustible Solids
WGK
WGK 3
闪点(°F)
Not applicable
闪点(°C)
Not applicable
个人防护装备
Eyeshields, Gloves, type N95 (US)
Joan E Nichols et al.
Virology, 534, 80-86 (2019-06-21)
The immunopathological mechanisms as well as the role played by influenza A virus infection of human leukocytes and induction of apoptosis have not been fully elucidated. We confirm here that the percentage of cells that are infected is less than
Ana Sofia Brito et al.
PLoS genetics, 16(2), e1008634-e1008634 (2020-02-19)
The dimorphic transition from the yeast to the filamentous form of growth allows cells to explore their environment for more suitable niches and is often crucial for the virulence of pathogenic fungi. In contrast to their Mep1/3 paralogues, fungal Mep2-type
Kristian Parey et al.
Science advances, 5(12), eaax9484-eaax9484 (2019-12-18)
Respiratory complex I is a redox-driven proton pump, accounting for a large part of the electrochemical gradient that powers mitochondrial adenosine triphosphate synthesis. Complex I dysfunction is associated with severe human diseases. Assembly of the one-megadalton complex I in the
Zhao Zhang et al.
Nature chemistry, 9(7), 653-659 (2017-06-24)
The diverse structure and regulated deformation of lipid bilayer membranes are among a cell's most fascinating features. Artificial membrane-bound vesicles, known as liposomes, are versatile tools for modelling biological membranes and delivering foreign objects to cells. To fully mimic the
Gabriela Camporeale et al.
Redox biology, 11, 38-50 (2016-11-20)
Infection with oncogenic human papillomavirus induces deregulation of cellular redox homeostasis. Virus replication and papillomavirus-induced cell transformation require persistent expression of viral oncoproteins E7 and E6 that must retain their functionality in a persistent oxidative environment. Here, we dissected the
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