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Merck

19-2930

Sigma-Aldrich

甲基纤维素

CP, viscosity 4000 cP 

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About This Item

CAS号:
MDL编号:
UNSPSC代码:
12352201

等级

CP

存货情况

available only in Japan

粘度

4000 cP

SMILES字符串

[*]OC[C@H]1O[C@@H](O[C@@H]2[C@@H](CO[*])O[C@@H](O[*])[C@H](O[*])[C@H]2O[*])[C@H](O[*])[C@@H](O[*])[C@@H]1O[*]

InChI key

YLGXILFCIXHCMC-JHGZEJCSSA-N

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储存分类代码

11 - Combustible Solids

WGK

WGK 1

闪点(°F)

Not applicable

闪点(°C)

Not applicable

个人防护装备

Eyeshields, Gloves, type N95 (US)


历史批次信息供参考:

分析证书(COA)

Lot/Batch Number

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Walter Fuchs et al.
Journal of virology, 83(8), 3930-3943 (2009-02-06)
Cleavage and encapsidation of newly replicated herpes simplex virus type 1 (HSV-1) DNA requires several essential viral gene products that are conserved in sequence within the Herpesviridae. However, conservation of function has not been analyzed in greater detail. For functional
Elsa-Noah N'Diaye et al.
EMBO reports, 10(2), 173-179 (2009-01-17)
Ubiquilins (UBQLNs) are adaptor proteins thought to deliver ubiquitinated substrates to proteasomes. Here, we show a role for UBQLN in autophagy: enforced expression of UBQLN protects cells from starvation-induced death, whereas depletion of UBQLN renders cells more susceptible. The UBQLN
Yihua Wang et al.
Biochemistry, 52(9), 1611-1621 (2013-02-07)
Myosin powers contraction in heart and skeletal muscle and is a leading target for mutations implicated in inheritable muscle diseases. During contraction, myosin transduces ATP free energy into the work of muscle shortening against resisting force. Muscle shortening involves relative
Hisham Al-Obaidi et al.
International journal of pharmaceutics, 443(1-2), 95-102 (2013-01-10)
Solid dispersions of varying weight ratios compositions of the nonionic drug, griseofulvin and the hydrophilic, anionic polymer, hydroxylpropylmethyl cellulose acetate succinate, have been prepared by ball milling and the resulting samples characterized using a combination of Fourier transform infra-red spectroscopy
Maryam Zadeh-Khorasani et al.
Journal of translational medicine, 11, 4-4 (2013-01-09)
Animal models of human inflammatory diseases have limited predictive quality for human clinical trials for various reasons including species specific activation mechanisms and the immunological background of the animals which markedly differs from the genetically heterogeneous and often aged patient

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