所有图片(1)
About This Item
线性分子式:
[CH3CH(OH)COO]2Ca · xH2O
CAS号:
分子量:
218.22 (anhydrous basis)
Beilstein:
4923285
EC號碼:
MDL號碼:
分類程式碼代碼:
12352207
PubChem物質ID:
NACRES:
NA.22
推荐产品
化驗
~90%
儲存溫度
2-8°C
SMILES 字串
O.C[C@H](O)C(=O)O[Ca]OC(=O)[C@H](C)O
InChI
1S/2C3H6O3.Ca.H2O/c2*1-2(4)3(5)6;;/h2*2,4H,1H3,(H,5,6);;1H2/q;;+2;/p-2/t2*2-;;/m00../s1
InChI 密鑰
MDAVASCOAJMZHZ-NVKWYWNSSA-L
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應用
Calcium L-lactate hydrate has been used as a sole carbon and energy source, to study the sulfate reduction ability of Desulfovibrio desulfuricans under different conditions.
儲存類別代碼
11 - Combustible Solids
水污染物質分類（WGK）
WGK 1
閃點（°F）
Not applicable
閃點（°C）
Not applicable
個人防護裝備
Eyeshields, Gloves, type N95 (US)
其他客户在看
Factors affecting microbial sulfate reduction by Desulfovibrio desulfuricans in continuous culture: limiting nutrients and sulfide concentration.
Okabe S, et al.
Biotechnology and Bioengineering, 40(6), 725-734 (1992)
Gaël Auray et al.
Frontiers in immunology, 11, 1429-1429 (2020-08-01)
The present study investigated the transcriptomic response of porcine dendritic cells (DC) to innate stimulation in vitro and in vivo. The aim was to identify DC subset-specialization, suitable Toll-like receptor (TLR) ligands targeting plasmacytoid DC (pDC), and the DC activation
Marlies P Noz et al.
Stroke, 49(12), 2910-2917 (2018-12-21)
Background and Purpose- Cerebral small vessel disease (cSVD) is the major vascular cause of cognitive decline and dementia. The pathogenesis of cSVD remains largely unknown, although several studies suggest a role for systemic inflammation. In certain pathophysiological situations, monocytes can
Effects of temperature and phosphorous concentration on microbial sulfate reduction by Desulfovibrio desulfuricans.
Okabe S and Characklis WG.
Biotechnology and Bioengineering, 39(10), 1031-1042 (1992)
Jorge Domínguez-Andrés et al.
Cell metabolism, 29(1), 211-220 (2018-10-09)
Sepsis involves simultaneous hyperactivation of the immune system and immune paralysis, leading to both organ dysfunction and increased susceptibility to secondary infections. Acute activation of myeloid cells induced itaconate synthesis, which subsequently mediated innate immune tolerance in human monocytes. In contrast, induction
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