化驗
99.7%
形狀
wire
自燃溫度
1198 °F
製造商/商標名
Goodfellow 679-663-85
電阻係數
4.46 μΩ-cm, 20°C
長度 × 直徑
0.3 m × 0.356 mm
bp
2970 °C (lit.)
mp
1278 °C (lit.)
密度
1.85 g/mL at 25 °C (lit.)
SMILES 字串
[Be]
InChI
1S/Be
InChI 密鑰
ATBAMAFKBVZNFJ-UHFFFAOYSA-N
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一般說明
For updated SDS information please visit www.goodfellow.com.
法律資訊
Product of Goodfellow
訊號詞
Danger
危險分類
Acute Tox. 3 Oral - Carc. 1B - Eye Irrit. 2 - Skin Irrit. 2 - Skin Sens. 1 - STOT RE 1
儲存類別代碼
6.1C - Combustible acute toxic Cat.3 / toxic compounds or compounds which causing chronic effects
水污染物質分類(WGK)
WGK 3
閃點(°F)
Not applicable
閃點(°C)
Not applicable
Current opinion in immunology, 25(6), 775-780 (2013-08-28)
Chronic beryllium disease (CBD) is a granulomatous lung disorder caused by a hypersensitivity to beryllium and characterized by the accumulation of beryllium-specific CD4(+) T cells in the lung. Genetic susceptibility to beryllium-induced disease is strongly associated with HLA-DP alleles possessing
Journal of environmental monitoring : JEM, 8(6), 598-604 (2006-06-13)
Standardized conventions governing the fractions of airborne particles that can penetrate the human head airways, the thoracic airways and the alveolar spaces have been internationally (although not universally) adopted. Several agencies involved in setting limit values for occupational exposure concentrations
Clinical immunology (Orlando, Fla.), 121(2), 134-143 (2006-05-16)
Chronic beryllium disease (CBD) is a granulomatous lung disorder caused by beryllium exposure in the workplace and is characterized by the accumulation of beryllium-specific CD4(+) T cells. Depending on genetic susceptibility and the nature of the exposure, CBD occurs in
Critical reviews in toxicology, 42(2), 107-118 (2012-01-27)
There is controversy on whether occupational exposure to beryllium causes lung cancer. We conducted a systematic review of epidemiologic studies on cancer among workers exposed to beryllium, including a study of seven U.S. production plants which has been recently updated
Cell, 158(1), 132-142 (2014-07-06)
T-cell-mediated hypersensitivity to metal cations is common in humans. How the T cell antigen receptor (TCR) recognizes these cations bound to a major histocompatibility complex (MHC) protein and self-peptide is unknown. Individuals carrying the MHCII allele, HLA-DP2, are at risk
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