所有图片(1)
About This Item
经验公式（希尔记法）:
C6H13N3O3
CAS号:
分子量:
175.19
Beilstein:
1725416
MDL號碼:
分類程式碼代碼:
12352209
PubChem物質ID:
推荐产品
化驗
98%
形狀
crystals
應用
peptide synthesis
SMILES 字串
N[C@@H](CCCNC(N)=O)C(O)=O
InChI
1S/C6H13N3O3/c7-4(5(10)11)2-1-3-9-6(8)12/h4H,1-3,7H2,(H,10,11)(H3,8,9,12)/t4-/m0/s1
InChI 密鑰
RHGKLRLOHDJJDR-BYPYZUCNSA-N
儲存類別代碼
11 - Combustible Solids
水污染物質分類（WGK）
WGK 3
閃點（°F）
Not applicable
閃點（°C）
Not applicable
個人防護裝備
Eyeshields, Gloves, type N95 (US)
P Crenn et al.
Gastroenterology, 119(6), 1496-1505 (2000-12-13)
No blood marker assessing the functional absorptive bowel length has been identified. Plasma citrulline, a nonprotein amino acid produced by intestinal mucosa, is one candidate. We tested this hypothesis in adult patients with the short-bowel syndrome, whose condition can lead
Cecilia Orellana et al.
Annals of the rheumatic diseases, 73(4), 752-755 (2013-07-28)
To study the impact of parity history on the risk of antibodies to citrullinated peptide antigens (ACPA) positive and ACPA-negative rheumatoid arthritis (RA), in different age-groups. Data from a population-based case-control study of female incident RA cases were analysed (2035
S Kaur et al.
Journal of dental research, 92(5), 399-408 (2013-03-26)
This systematic review considers the evidence available for a relationship between periodontal disease and rheumatoid arthritis. MEDLINE/PubMed, CINAHL, DOSS, Embase, Scopus, Web of Knowledge, MedNar, and ProQuest Theses and Dissertations were searched from the inception of the database until June
C C Reparon-Schuijt et al.
Arthritis and rheumatism, 44(1), 41-47 (2001-02-24)
To understand the regulation of anti-citrulline-containing peptide antibody (anti-CCP) production in rheumatoid arthritis (RA), production of anti-CCP by B cells derived from peripheral blood (PB), bone marrow (BM), and synovial fluid (SF) was examined. Purified PB and SF B cells
Koen M J Janssen et al.
Current opinion in rheumatology, 25(2), 241-247 (2013-02-02)
This article reviews the link between periodontitis and rheumatoid arthritis (RA) with regard to similarities in genetic risk factors and immunopathogenesis. Emphasis is paid to the potential role of the periodontal pathogen Porphyromonas gingivalis in the etiopathogenesis of both periodontitis
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