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蒸汽密度
4.8 (vs air)
蒸汽壓力
0.38 mmHg ( 20 °C)
1 mmHg ( 42 °C)
化驗
95%
折射率
n20/D 1.497 (lit.)
bp
198-199 °C (lit.)
mp
23-26 °C (lit.)
密度
0.885 g/mL at 25 °C (lit.)
SMILES 字串
C\C(C)=C\C(=O)\C=C(\C)C
InChI
1S/C9H14O/c1-7(2)5-9(10)6-8(3)4/h5-6H,1-4H3
InChI 密鑰
MTZWHHIREPJPTG-UHFFFAOYSA-N
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儲存類別代碼
10 - Combustible liquids
水污染物質分類(WGK)
WGK 3
閃點(°F)
174.2 °F - closed cup
閃點(°C)
79 °C - closed cup
個人防護裝備
Eyeshields, Gloves, multi-purpose combination respirator cartridge (US)
Journal of hepatology, 46(5), 858-868 (2007-02-06)
The agonistic Jo2 anti-Fas antibody reproduces human fulminant hepatitis in mice. We tested the hypothesis that enhancing hepatic glutathione (GSH) stores may prevent Jo2-induced apoptosis. We fed mice with a normal diet or a sulfur amino acid-enriched (SAA(+)) diet increasing
Biochemical and biophysical research communications, 317(1), 149-156 (2004-03-30)
Using isolated rat hepatocytes we have shown that glutathione (GSH) depletion by glutathione-S-transferase (GST)-catalyzed conjugation with 1-bromoheptane or phorone was accompanied by a significant elevation in ascorbate synthesis. Glycogenolysis was also stimulated without a significant rise in glucose synthesis. Furthermore
Free radical biology & medicine, 38(7), 867-873 (2005-03-08)
Most animals synthesize ascorbate. It is an essential enzymatic cofactor for the synthesis of a variety of biological molecules and also a powerful antioxidant. There is, however, little direct evidence supporting an antioxidant role for endogenously produced ascorbate. Recently, we
Chemico-biological interactions, 128(2), 97-113 (2000-10-12)
Previously we reported that methylsulphonyl-2,6-dichlorobenzene, 2, 6-(diCl-MeSO(2)-B), was irreversibly bound to the olfactory mucosa of mice and induced necrosis of the Bowman's glands with subsequent neuroepithelial degeneration and detachment. In this study, autoradiography and histopathology were used to determine tissue-localization
Hepatology (Baltimore, Md.), 36(1), 55-64 (2002-06-27)
The effect of reduced glutathione (GSH) depletion by acetaminophen (APAP), diethylmaleate (DEM), or phorone on the mode of cell death and susceptibility to tumor necrosis factor (TNF)-induced cell death was studied in cultured mouse hepatocytes. Dose-dependent necrosis was the exclusive
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