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AP136F

Sigma-Aldrich

Goat Anti-Rat IgG Antibody, FITC conjugate

26 mg/mL, Chemicon®

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About This Item

Code UNSPSC :
12352203
eCl@ss :
32160702
Nomenclature NACRES :
NA.46

Source biologique

goat

Niveau de qualité

Conjugué

FITC conjugate

Forme d'anticorps

F(ab′)2 fragment of affinity isolated antibody

Type de produit anticorps

secondary antibodies

Clone

polyclonal

Espèces réactives

rat

Fabricant/nom de marque

Chemicon®

Concentration

26 mg/mL

Technique(s)

immunofluorescence: suitable

Conditions d'expédition

wet ice

Modification post-traductionnelle de la cible

unmodified

Spécificité

Reacts with Rat IgG gamma chain, as well as the light chains from all rat immunoglobulin classes. Monospecific on IEP against whole rat serum. Single band on IEP at a concentration of 10 mg/mL when run against rabbit anti goat whole serum.

FLUOROPHORE/ PROTEIN:

Approximately 12 mg/mg.

Immunogène

Highly purified Rat IgG isolated from pooled normal rat serum.

Application

Goat anti-Rat IgG Antibody, FITC conjugate is an antibody against Rat IgG for use in IF.
Research Category
Secondary & Control Antibodies
Research Sub Category
Whole Immunoglobulin Secondary Antibodies

Stockage et stabilité

When protected from light, the lyophilized antibody conjugate is stable at 2-8°C.

DO NOT FREEZE.

Informations légales

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

Clause de non-responsabilité

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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Mentions de danger

Conseils de prudence

Classification des risques

Aquatic Chronic 3

Code de la classe de stockage

11 - Combustible Solids

Classe de danger pour l'eau (WGK)

WGK 3


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Gioia Marino et al.
Science advances, 9(28), eadh1403-eadh1403 (2023-07-14)
Intensive physical activity improves motor functions in patients with Parkinson's disease (PD) at early stages. However, the mechanisms underlying the beneficial effects of exercise on PD-associated neuronal alterations have not been fully clarified yet. Here, we tested the hypothesis that
Zheng Liu et al.
Redox biology, 52, 102305-102305 (2022-04-04)
Hepatocellular cell death and macrophage proinflammatory activation contribute to the pathology of various liver diseases, during which XBP1 plays an important role. However, the function and mechanism of XBP1 in thioacetamide (TAA)-induced acute liver injury (ALI) remains unknown. Here, we
Shintaro Nakao et al.
The American journal of pathology, 171(3), 1058-1065 (2007-08-11)
Dexamethasone, a synthetic corticosteroid, is widely used as a potent anti-inflammatory drug in various diseases including corneal angiogenesis. However, dexamethasone's impact on interleukin (IL)-1beta-dependent inflammatory angiogenesis is unknown. Here, we show that dexamethasone inhibits IL-1beta-induced neovascularization and the expression of
Alessandro Tozzi et al.
Brain : a journal of neurology, 144(11), 3477-3491 (2021-07-24)
Misfolding and aggregation of α-synuclein are specific features of Parkinson's disease and other neurodegenerative diseases defined as synucleinopathies. Parkinson's disease progression has been correlated with the formation and extracellular release of α-synuclein aggregates, as well as with their spread from
J K L Woodward et al.
Eye (London, England), 19(3), 342-348 (2004-07-20)
To establish if invasive and noninvasive uveal melanomas have differences in expression of adhesion molecules, and whether their adhesive interactions with the extracellular matrix (ECM) and endothelium vary. Cells from an invasive and noninvasive uveal melanoma cell line and hepatic

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