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Merck
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792519

Sigma-Aldrich

Bicarbonato de sodio

anhydrous, free-flowing, Redi-Dri™, ACS reagent, ≥99.7%

Sinónimos:

Sodium hydrogen carbonate

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About This Item

Fórmula lineal:
NaHCO3
Número de CAS:
144-55-8
Peso molecular:
84.01
Beilstein:
4153970
Número CE:
205-633-8
Número MDL:
MFCD00003528
Código UNSPSC:
12352302
ID de la sustancia en PubChem:
329768339
NACRES:
NA.21

grado

ACS reagent
anhydrous

Nivel de calidad

100

Agency

suitable for EPA 1621
suitable for EPA 300

Línea del producto

Redi-Dri™

Análisis

≥99.7%

formulario

powder

calidad

free-flowing

impurezas

≤0.015% insoluble matter

pKa (25 °C)

(1) 6.37, (2) 10.25 (carbonic acid)

densidad

2.16 g/mL at 25 °C (lit.)

trazas de anión

chloride (Cl-): ≤0.003%
phosphate (PO43-): ≤0.001 ppm
sulfur compounds (as SO42-): ≤0.003%

trazas de catión

Ca: ≤0.02%
Fe: ≤0.001%
K: ≤0.005%
Mg: ≤0.005%
NH4+: ≤5 ppm
heavy metals: ≤5 ppm (by ICP-OES)

cadena SMILES

[Na+].OC([O-])=O

InChI

1S/CH2O3.Na/c2-1(3)4;/h(H2,2,3,4);/q;+1/p-1

Clave InChI

UIIMBOGNXHQVGW-UHFFFAOYSA-M

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Descripción general

Sodium bicarbonate is commonly known as baking soda. It serves as a source of carbon dioxide and is used to prepare a variety of sodium salts. It is also an important pH-regulating agent.

Aplicación

Sodium bicarbonate can be used as a reactant to synthesize monosodium Schiff bases by the condensation reaction of aldehydes and amino acids.

Características y beneficios

  • Increased efficiency – Eliminates time and effort of loosening hardened powders      
  • Safety – Eliminates tools of force used to break up clumps
  • Ecology – Reduces waste as it does not stick to the packaging
  • Economy – Faster preparation and solvation increase productivity and reduce costs     
  • Assured Quality – Excellent, expert-tested quality with no anti-caking agents
  • Flexible volumes – Available from research amounts to scale-up quantities

Información legal

Redi-Dri is a trademark of Sigma-Aldrich Co. LLC

Código de clase de almacenamiento

11 - Combustible Solids

Clase de riesgo para el agua (WGK)

WGK 1

Punto de inflamabilidad (°F)

Not applicable

Punto de inflamabilidad (°C)

Not applicable

Certificados de análisis (COA)

Busque Certificados de análisis (COA) introduciendo el número de lote del producto. Los números de lote se encuentran en la etiqueta del producto después de las palabras «Lot» o «Batch»

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Joshua C Tremblay et al.
The Journal of physiology, 593(3), 723-737 (2014-11-25)
Blood flow through intrapulmonary arteriovenous anastomoses (IPAVA) is increased by acute hypoxia during rest by unknown mechanisms. Oral administration of acetazolamide blunts the pulmonary vascular pressure response to acute hypoxia, thus permitting the observation of IPAVA blood flow with minimal
Qian Liang et al.
Electrophoresis, 36(5), 731-736 (2014-11-26)
Although the existing theories have predicted enhancement of electrophoretic mobility of microparticles near a solid wall, the relevant experimental studies are rare. This is mainly due to difficulties in experimentally controlling and measuring particle-wall separations under dynamic electrophoretic conditions. This
Kate M Hatzopoulos et al.
Investigative ophthalmology & visual science, 55(6), 3957-3970 (2014-06-05)
Retinal angiogenesis, neural dysfunction, and microglial activation occur in both humans with retinopathy of prematurity and in animal models of the disease. The aim of this study was to assess whether blockade of the renin-angiotensin system ameliorated these effects in
Roberto J Diaz et al.
American journal of physiology. Cell physiology, 306(12), C1191-C1199 (2014-04-25)
We have previously shown that ischemic preconditioning (IPC) protection against necrosis in whole hearts and in both fresh and cultured cardiomyocytes, as well as the improved regulatory volume decrease to hypoosmotic swelling in cardiomyocytes, is abrogated through Cl(-) channel blockade
Erik van Kampen et al.
Journal of leukocyte biology, 96(5), 833-841 (2014-07-16)
Alterations in DNA methylation patterns in peripheral blood leukocytes precede atherosclerotic lesion development in mouse models of atherosclerosis and have been linked to cardiovascular death in patients. The aim of this study is to investigate the long-term changes induced by
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