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EMU090361

Sigma-Aldrich

MISSION® esiRNA

targeting mouse Timp2

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About This Item

UNSPSC Code:
41105324
NACRES:
NA.51

description

Powered by Eupheria Biotech

product line

MISSION®

form

lyophilized powder

esiRNA cDNA target sequence

ATCTATGGCAACCCCATCAAGAGGATTCAGTATGAGATCAAGCAGATAAAGATGTTCAAAGGACCTGACAAAGACATCGAGTTTATCTACACGGCCCCCTCTTCAGCAGTGTGCGGGGTCTCGCTGGACGTTGGAGGAAAGAAGGAGTATCTAATTGCAGGAAAGGCAGAAGGAGATGGCAAGATGCACATTACCCTCTGTGACTTCATTGTGCCCTGGGACACGCTTAGCATCACCCAGAAGAAGAGCCTGAACCACAGGTACCAGATGGGCTGTGAGTGCAAGATCACTCGCTGTCCCATGATCCCTTGCTACATCTCCTCCCCGGATGAGTGCCTCTGGATGGACTGGGTCACAGAGAAGAGCATCAATGGGCACCAGGCCAAGTTCTTCGCCTGCATCAAGAGAAGTGATGGTTCTTGCGCGTGGTACCGCGGGGCGGCACCCCCCAAGCAAGAGTTTCTTGACATCGAGGACCCGTAAGAAGGCTGACAGAGCCCCTGTGGCCAATTGAAAAGCCTCTGAGGGTTTAGACTGGTCCAGCTTTGACATCCCTTCCTGGA

Ensembl | mouse accession no.

NCBI accession no.

shipped in

ambient

storage temp.

−20°C

Gene Information

General description

MISSION® esiRNA are endoribonuclease prepared siRNA. They are a heterogeneous mixture of siRNA that all target the same mRNA sequence. These multiple silencing triggers lead to highly-specific and effective gene silencing.

For additional details as well as to view all available esiRNA options, please visit SigmaAldrich.com/esiRNA.

Legal Information

MISSION is a registered trademark of Merck KGaA, Darmstadt, Germany

Storage Class Code

10 - Combustible liquids

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable


Certificates of Analysis (COA)

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An Yan et al.
Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology, 37(1), 55-66 (2015-08-19)
The aim of this study was to investigate the role of microRNA miR-761 in the progression and metastasis of non-small cell lung cancer (NSCLC), and the mechanisms by which miR-761 regulates cell proliferation and metastatic activity of NSCLC cell lines.
Qinhong Xu et al.
Oncotarget, 6(16), 14153-14164 (2015-04-18)
MicroRNAs are involved in the initiation and progression of pancreatic cancer. In this study, we showed that miR-221/222 is overexpressed in pancreatic cancer. MiR-221/222 overexpression significantly promoted pancreatic cancer cell proliferation and invasion while inhibiting apoptosis. The expression of the

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