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Key Documents

MAB5478

Sigma-Aldrich

Anti-Retinoid X Receptor α Antibody

ascites fluid, Chemicon®

Synonym(s):

RXRalpha

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About This Item

UNSPSC Code:
12352203
eCl@ss:
32160702
NACRES:
NA.41

biological source

mouse

antibody form

ascites fluid

antibody product type

primary antibodies

clone

monoclonal

species reactivity

human, mouse

manufacturer/tradename

Chemicon®

technique(s)

ELISA: suitable
electrophoretic mobility shift assay: suitable
immunoprecipitation (IP): suitable
western blot: suitable

isotype

IgG1

NCBI accession no.

NM_002957.3

UniProt accession no.

P19793

shipped in

wet ice

target post-translational modification

unmodified

Gene Information

human ... RXRA(6256)

Specificity

Reacts with Retinoid X Receptor alpha (RXRalpha). No cross reactivity with RXRbeta or RXRgamma.

Immunogen

Fusion protein corresponding to the DE-region of mouse RXRa.

Application

Use Anti-Retinoid X Receptor α Antibody (Mouse Monoclonal Antibody) validated in ELISA, EMSA, IP, WB to detect Retinoid X Receptor α also known as RXR alpha.
Western blot (endogenous and recombinant protein): 1:500-1:5,000

Immunocytochemistry (transfected cells): 1:500-1:5,000

Immunoprecipitation (endogenous and recombinant protein): 1:500-1:5,000

Gel shift (endogenous and recombinant protein): 1:500-1:5,000

ELISA: 1:500-1:5,000

Optimal working dilutions must be determined by end user.

Legal Information

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

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Storage Class Code

10 - Combustible liquids

WGK

WGK 1

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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Retinoid-X-receptors (?/?) in melanocytes modulate innate immune responses and differentially regulate cell survival following UV irradiation.
Coleman, DJ; Garcia, G; Hyter, S; Jang, HS; Chagani, S; Liang, X; Larue, L; Ganguli-Indra et al.
PLoS Genetics null

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