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MAB1582

Sigma-Aldrich

Anti-Chondroitin Sulfate Proteoglycan Antibody, carbohydrate epitope, clone Cat-316

ascites fluid, clone Cat-316, Chemicon®

Synonym(e):

CSPG

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About This Item

UNSPSC-Code:
12352203
eCl@ss:
32160702
NACRES:
NA.41

Biologische Quelle

mouse

Qualitätsniveau

100

Antikörperform

ascites fluid

Antikörper-Produkttyp

primary antibodies

Klon

Cat-316, monoclonal

Speziesreaktivität

feline, rat

Hersteller/Markenname

Chemicon®

Methode(n)

immunocytochemistry: suitable
immunohistochemistry: suitable
immunoprecipitation (IP): suitable
western blot: suitable

Isotyp

IgM

Versandbedingung

dry ice

Posttranslationale Modifikation Target

unmodified

Angaben zum Gen

rat ... Cspg4(81651)

Spezifität

Chondroitin sulfate proteoglycan (CSPG), carbohydrate epitope

Immunogen

Epitope: carbohydrate epitope
Feline brain proteoglycans

Anwendung

Research Category
Zellstruktur
Research Sub Category
Integrine
Anti-Chondroitin Sulfate Proteoglycan Antibody, carbohydrate epitope, clone Cat-316 is an antibody against Chondroitin Sulfate Proteoglycan for use in IP, WB, IC, IH.
Immunohistochemistry: 1:70,000 on 4% paraformaldehyde fixed frozen tissue.

Immunocytochemistry: 1:2,500 on primary cultures of neurons.

Immunoblot: 1:200,000

Immunoprecipitation

Optimal working dilutions must be determined by the end user.

Physikalische Form

Liquid with 0.1% sodium azide.

Lagerung und Haltbarkeit

Maintain at -20°C in undiluted aliquots for up to 6 months. Avoid repeated freeze/thaw cycles

Rechtliche Hinweise

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

Haftungsausschluss

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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Lagerklassenschlüssel

12 - Non Combustible Liquids

WGK

nwg

Flammpunkt (°F)

Not applicable

Flammpunkt (°C)

Not applicable

Analysenzertifikate (COA)

Suchen Sie nach Analysenzertifikate (COA), indem Sie die Lot-/Chargennummer des Produkts eingeben. Lot- und Chargennummern sind auf dem Produktetikett hinter den Wörtern ‘Lot’ oder ‘Batch’ (Lot oder Charge) zu finden.

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Die Dokumentenbibliothek aufrufen

Satomi Nadanaka et al.
Biomolecules, 10(11) (2020-11-05)
The chondroitin sulfate (CS)-rich dense extracellular matrix surrounding neuron cell bodies and proximal dendrites in a mesh-like structure is called a perineuronal net (PNN). CS chains in PNNs control neuronal plasticity by binding to PNN effectors, semaphorin-3A (Sema3A) and orthodenticle
De-Lai Zhao et al.
Molecular medicine reports, 22(4), 2637-2644 (2020-09-19)
Chondrocytes in injured cartilage tissue are susceptible to mechanical loading; mechanical overloading can induce cartilage degeneration. The aim of the present study was to investigate whether mechanical loading can regulate chondrocyte degeneration and angiogenesis via the tissue inhibitor of matrix
Shinji Miyata et al.
Frontiers in integrative neuroscience, 12, 3-3 (2018-02-20)
Aggrecan, a chondroitin sulfate (CS) proteoglycan, forms lattice-like extracellular matrix structures called perineuronal nets (PNNs). Neocortical PNNs primarily ensheath parvalbumin-expressing inhibitory neurons (parvalbumin, PV cells) late in brain development. Emerging evidence indicates that PNNs promote the maturation of PV cells
Hiroshi Ueno et al.
IBRO reports, 4, 22-37 (2018-08-24)
Specific regions of the cerebral cortex are highly plastic in an organism's lifetime. It is thought that perineuronal nets (PNNs) regulate plasticity, but labeling for Wisteria floribunda agglutinin (WFA), which is widely used to detect PNNs, is observed throughout the
Guixin Zhang et al.
The Journal of comparative neurology, 522(9), 2209-2229 (2013-12-21)
Disability following spinal cord injury is due to failure of axon regeneration, which has been ascribed to environmental factors in the central nervous system and a developmental loss of intrinsic growth capacity in neurons. Recently, the receptor-like protein tyrosine phosphatases

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